Rapid induction of heparin-binding epidermal growth factor/diphtheria toxin receptor expression by Raf and Ras oncogenes.
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PI-3 kinase activity is necessary for ERK1/2-induced disruption of mammary epithelial architecture.Direct activation of the stress-activated protein kinase (SAPK) and extracellular signal-regulated protein kinase (ERK) pathways by an inducible mitogen-activated protein Kinase/ERK kinase kinase 3 (MEKK) derivativeRaf-1 physically interacts with Rb and regulates its function: a link between mitogenic signaling and cell cycle regulationRoles of the Raf/MEK/ERK pathway in cell growth, malignant transformation and drug resistanceRETRACTED: Inactivation of MAP kinase signalling in Myc transformed cells and rescue by LiCl inhibition of GSK3.Heparin-binding epidermal growth factor-like growth factor/diphtheria toxin receptor in normal and neoplastic hematopoiesisERK5 and ERK2 cooperate to regulate NF-kappaB and cell transformationRAS oncogenes: weaving a tumorigenic webInduction of cyclooxygenase-2 by activated Ha-ras oncogene in Rat-1 fibroblasts and the role of mitogen-activated protein kinase pathwayHeparin-binding epidermal-growth-factor-like growth factor gene expression is induced by scrape-wounding epithelial cell monolayers: involvement of mitogen-activated protein kinase cascadesFrom bench to bedside: lessons learned in translating preclinical studies in cancer drug developmentIdentification of a novel ligand-receptor pair constitutively activated by ras oncogenesRapid and bi-directional regulation of AMPA receptor phosphorylation and trafficking by JNKEndo180 binds to the C-terminal region of type I collagenConditionally oncogenic forms of the A-Raf and B-Raf protein kinases display different biological and biochemical properties in NIH 3T3 cellsState-dependent Ras signaling and AMPA receptor trafficking.Skeletal muscle cell activation by low-energy laser irradiation: a role for the MAPK/ERK pathway.Cross-cascade activation of ERKs and ternary complex factors by Rho family proteins.Induction of postmitotic neuroretina cell proliferation by distinct Ras downstream signaling pathways.Induction of beta3-integrin gene expression by sustained activation of the Ras-regulated Raf-MEK-extracellular signal-regulated kinase signaling pathway.Loss of EGFR signaling regulated miR-203 promotes prostate cancer bone metastasis and tyrosine kinase inhibitors resistance.The stress inducer arsenite activates mitogen-activated protein kinases extracellular signal-regulated kinases 1 and 2 via a MAPK kinase 6/p38-dependent pathway.The Ras/Raf/MEK/extracellular signal-regulated kinase pathway induces autocrine-paracrine growth inhibition via the leukemia inhibitory factor/JAK/STAT pathway.Analysis of the transcriptional program induced by Raf in epithelial cellsSignal transduction mediated by the Ras/Raf/MEK/ERK pathway from cytokine receptors to transcription factors: potential targeting for therapeutic intervention.CEMP1 Induces Transformation in Human Gingival Fibroblasts.Raf induces NF-kappaB by membrane shuttle kinase MEKK1, a signaling pathway critical for transformation.The transcriptional response to Raf activation is almost completely dependent on Mitogen-activated Protein Kinase Kinase activity and shows a major autocrine component.The MAPK MEK1/2-ERK1/2 Pathway and Its Implication in Hepatocyte Cell Cycle Control.Heparin-binding epidermal growth factor-like growth factor, a v-Jun target gene, induces oncogenic transformation.Raf, but not MEK or ERK, is sufficient for differentiation of hippocampal neuronal cells.Activation of the mitogen-activated protein kinase pathway induces transcription of the PAC-1 phosphatase gene.Induction of cell proliferation in quiescent NIH 3T3 cells by oncogenic c-Raf-1.Raf-induced proliferation or cell cycle arrest is determined by the level of Raf activity with arrest mediated by p21Cip1.Real-time imaging reveals that noninvasive mammary epithelial acini can contain motile cells.ERK1/2 mediate wounding- and G-protein-coupled receptor ligands-induced EGFR activation via regulating ADAM17 and HB-EGF shedding.Oncogenic BRAF(V600E) inhibits BIM expression to promote melanoma cell survival.TRIM11 is overexpressed in high-grade gliomas and promotes proliferation, invasion, migration and glial tumor growth.Sustained activation of Ras/Raf/mitogen-activated protein kinase cascade by the tumor suppressor p53.Production of heparin binding epidermal growth factor-like growth factor in the early phase of regeneration after acute renal injury. Isolation and localization of bioactive molecules.
P2860
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P2860
Rapid induction of heparin-binding epidermal growth factor/diphtheria toxin receptor expression by Raf and Ras oncogenes.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Rapid induction of heparin-bin ...... sion by Raf and Ras oncogenes.
@en
type
label
Rapid induction of heparin-bin ...... sion by Raf and Ras oncogenes.
@en
prefLabel
Rapid induction of heparin-bin ...... sion by Raf and Ras oncogenes.
@en
P2093
P356
P1433
P1476
Rapid induction of heparin-bin ...... ssion by Raf and Ras oncogenes
@en
P2093
J A Abraham
M L Samuels
S A McCarthy
P304
P356
10.1101/GAD.9.16.1953
P577
1995-08-01T00:00:00Z