c-Fos is required for excision repair of UV-light induced DNA lesions by triggering the re-synthesis of XPF
about
Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptationTranscriptional and Posttranslational Regulation of Nucleotide Excision Repair: The Guardian of the Genome against Ultraviolet RadiationCC3/TIP30 affects DNA damage repair.Non-catalytic Roles for XPG with BRCA1 and BRCA2 in Homologous Recombination and Genome StabilityAdaptive upregulation of DNA repair genes following benzo(a)pyrene diol epoxide protects against cell death at the expense of mutations.Testicular nuclear receptor 4 (TR4) regulates UV light-induced responses via Cockayne syndrome B protein-mediated transcription-coupled DNA repairGenotoxic stress inhibits Ewing sarcoma cell growth by modulating alternative pre-mRNA processing of the RNA helicase DHX9.Apoptosis induced by temozolomide and nimustine in glioblastoma cells is supported by JNK/c-Jun-mediated induction of the BH3-only protein BIM.Delayed c-Fos activation in human cells triggers XPF induction and an adaptive response to UVC-induced DNA damage and cytotoxicity.Three prime exonuclease I (TREX1) is Fos/AP-1 regulated by genotoxic stress and protects against ultraviolet light and benzo(a)pyrene-induced DNA damage.A sensitive flow cytometry-based nucleotide excision repair assay unexpectedly reveals that mitogen-activated protein kinase signaling does not regulate the removal of UV-induced DNA damage in human cells.Rac1 promotes diethylnitrosamine (DEN)-induced formation of liver tumors.Cdt2-mediated XPG degradation promotes gap-filling DNA synthesis in nucleotide excision repair.Late activation of stress-activated protein kinases/c-Jun N-terminal kinases triggered by cisplatin-induced DNA damage in repair-defective cells.The p38 mitogen-activated protein kinase augments nucleotide excision repair by mediating DDB2 degradation and chromatin relaxation.MKP-1 suppresses PARP-1 degradation to mediate cisplatin resistance.
P2860
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P2860
c-Fos is required for excision repair of UV-light induced DNA lesions by triggering the re-synthesis of XPF
description
2006 nî lūn-bûn
@nan
2006年の論文
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2006年論文
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2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
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2006年论文
@zh-cn
name
c-Fos is required for excision ...... gering the re-synthesis of XPF
@en
type
label
c-Fos is required for excision ...... gering the re-synthesis of XPF
@en
prefLabel
c-Fos is required for excision ...... gering the re-synthesis of XPF
@en
P2093
P2860
P356
P1476
c-Fos is required for excision ...... gering the re-synthesis of XPF
@en
P2093
Bernd Kaina
Dorthe Aasland
Judith Origer
Maja T Tomicic
Markus Christmann
P2860
P304
P356
10.1093/NAR/GKL895
P407
P577
2006-11-27T00:00:00Z