Perinuclear mitochondrial clustering creates an oxidant-rich nuclear domain required for hypoxia-induced transcription - Wikidata - awgldk - TriplyDB

Perinuclear mitochondrial clustering creates an oxidant-rich nuclear domain required for hypoxia-induced transcription

Perinuclear mitochondrial clustering creates an oxidant-rich nuclear domain required for hypoxia-induced transcription

http://www.wikidata.org/entity/Q38323862

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The Role of Mitochondrial Reactive Oxygen Species in Cardiovascular Injury and Protective Strategies Cross Talk of Proteostasis and Mitostasis in Cellular Homeodynamics, Ageing, and Disease Redox biology in pulmonary arterial hypertension (2013 Grover Conference Series)Cancer metabolism and oxidative stress: Insights into carcinogenesis and chemotherapy via the non-dihydrofolate reductase effects of methotrexate Hypoxia signaling pathways: modulators of oxygen-related organelles Regulation of mitochondrial biogenesis and its intersection with inflammatory responses Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioning Mitohormesis Beyond oxidative stress: an immunologist's guide to reactive oxygen species Targeting the redox balance in inflammatory skin conditions Dynamin-related protein 1 controls the migration and neuronal differentiation of subventricular zone-derived neural progenitor cells Mitochondria localize to the cleavage furrow in mammalian cytokinesis Optogenetic control of molecular motors and organelle distributions in cells.Muscle-specific Drp1 overexpression impairs skeletal muscle growth via translational attenuation.ROS homeostasis and metabolism: a dangerous liason in cancer cells Mitochondria in lung disease Aging-dependent changes in rat heart mitochondrial glutaredoxins--Implications for redox regulation ROS function in redox signaling and oxidative stress Physiological roles of mitochondrial reactive oxygen species MARCH5-mediated quality control on acetylated Mfn1 facilitates mitochondrial homeostasis and cell survival.Mitochondria, endothelial cell function, and vascular diseases.Microtubules and their role in cellular stress in cancer The cancer theory of pulmonary arterial hypertension.Uncoupling of the Hippo and Rho pathways allows megakaryocytes to escape the tetraploid checkpoint.Nuclear cytoplasmic trafficking of proteins is a major response of human fibroblasts to oxidative stress The ubiquitin-conjugating enzyme UBE2E3 and its import receptor importin-11 regulate the localization and activity of the antioxidant transcription factor NRF2.The Uve1 endonuclease is regulated by the white collar complex to protect cryptococcus neoformans from UV damage.Blood transfusion products contain mitochondrial DNA damage-associated molecular patterns: a potential effector of transfusion-related acute lung injury.The "fast" and the "slow" modes of mitochondrial DNA degradation.Mitochondria as signaling organelles.Mitophagy and cancer.Hypoxia-induced gene expression results from selective mRNA partitioning to the endoplasmic reticulum.Regulation of nuclear NF-κB oscillation by a diffusion coefficient and its biological implications Regulation of NF-κB Oscillation by Nuclear Transport: Mechanisms Determining the Persistency and Frequency of Oscillation Impaired mitophagy leads to cigarette smoke stress-induced cellular senescence: implications for chronic obstructive pulmonary disease DNA damage induces nuclear actin filament assembly by Formin -2 and Spire-½ that promotes efficient DNA repair. [corrected].Environmental Factors Can Influence Mitochondrial Inheritance in the Saccharomyces Yeast Hybrids An oxidative DNA "damage" and repair mechanism localized in the VEGF promoter is important for hypoxia-induced VEGF mRNA expression.MITOCHONDRIAL FUNCTION IN SEPSIS.Cancer-associated oxidoreductase ERO1-α drives the production of VEGF via oxidative protein folding and regulating the mRNA level.

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Perinuclear mitochondrial clustering creates an oxidant-rich nuclear domain required for hypoxia-induced transcription

http://www.wikidata.org/entity/Q38323862

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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Perinuclear mitochondrial clus ...... hypoxia-induced transcription

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Perinuclear mitochondrial clus ...... hypoxia-induced transcription

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Perinuclear mitochondrial clus ...... hypoxia-induced transcription

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Science Signaling

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Perinuclear mitochondrial clus ...... hypoxia-induced transcription

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P2093

Abu-Bakr Al-Mehdi

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Brad M Swiger

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Darla J Reed

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Gina C Bardwell

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Mark N Gillespie

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Mikhail F Alexeyev

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Mita R Patel

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Viktor M Pastukh

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Viktoriya V Pastukh

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P2860

Overexpression of the human MNB/DYRK1A gene induces formation of multinucleate cells through overduplication of the centrosome

Investigating mitochondrial redox potential with redox-sensitive green fluorescent protein indicators

VEGF signaling inside vascular endothelial cells and beyond.

Ca(2+) homeostasis during mitochondrial fragmentation and perinuclear clustering induced by hFis1

Ref-1/Ape is critical for formation of the hypoxia-inducible transcriptional complex on the hypoxic response element of the rat pulmonary artery endothelial cell VEGF gene

Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

Reactive oxygen species generated at mitochondrial complex III stabilize hypoxia-inducible factor-1alpha during hypoxia: a mechanism of O2 sensing

Differential mitochondrial distribution in human pronuclear embryos leads to disproportionate inheritance between blastomeres: relationship to microtubular organization, ATP content and competence.

HIF-1α stabilization by mitochondrial ROS promotes Met-dependent invasive growth and vasculogenic mimicry in melanoma cells.

Controlled DNA "damage" and repair in hypoxic signaling

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