Ternary complex factor-serum response factor complex-regulated gene activity is required for cellular proliferation and inhibition of apoptotic cell death.
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Disease progression mediated by egr-1 associated signaling in response to oxidative stressThe cardiac transcription network modulated by Gata4, Mef2a, Nkx2.5, Srf, histone modifications, and microRNAsRequirement of 3-phosphoinositide-dependent protein kinase-1 for BDNF-mediated neuronal survivalSerum response factor is required for cell contact maintenance but dispensable for proliferation in visceral yolk sac endotheliumTranscription factors Elk-1 and SRF are engaged in IL1-dependent regulation of ZC3H12A expression.Degeneration of the mouse retina upon dysregulated activity of serum response factor.Neurokinin B induces c-fos transcription via protein kinase C and activation of serum response factor and Elk-1 in immortalized GnRH neurons.Regulation of serum response factor activity and smooth muscle cell apoptosis by chromodomain helicase DNA-binding protein 8.Net expression inhibits the growth of pancreatic ductal adenocarcinoma cell PL45 in vitro and in vivoHeteromerization of GPR55 and cannabinoid CB2 receptors modulates signallingEpidermal growth factor regulates Mcl-1 expression through the MAPK-Elk-1 signalling pathway contributing to cell survival in breast cancerELK4 neutralization sensitizes glioblastoma to apoptosis through downregulation of the anti-apoptotic protein Mcl-1MCL-1, BCL-XL and MITF Are Diversely Employed in Adaptive Response of Melanoma Cells to Changes in Microenvironment.Dual Specificity Phosphatase 5, a Specific Negative Regulator of ERK Signaling, Is Induced by Serum Response Factor and Elk-1 Transcription FactorDownregulated protein expression of transcriptional activator ELK-1 in atrial myocardium of chronic AF patientsSignal-induced functions of the transcription factor TFII-I.Adhesion regulates MAP kinase/ternary complex factor exchange to control a proliferative transcriptional switchMouse tissues that undergo neoplastic progression after K-Ras activation are distinguished by nuclear translocation of phospho-Erk1/2 and robust tumor suppressor responses.Immunological synapse: a multi-protein signalling cellular apparatus for controlling gene expression.That which does not kill me makes me stronger; combining ERK1/2 pathway inhibitors and BH3 mimetics to kill tumour cells and prevent acquired resistance.An Elk transcription factor is required for Runx-dependent survival signaling in the sea urchin embryo.Mitogen-induced recruitment of ERK and MSK to SRE promoter complexes by ternary complex factor Elk-1.HCMV activation of ERK-MAPK drives a multi-factorial response promoting the survival of infected myeloid progenitors.Relationship between serum response factor and androgen receptor in prostate cancer.The kinase LMTK3 promotes invasion in breast cancer through GRB2-mediated induction of integrin β₁.SRF Co-factors Control the Balance between Cell Proliferation and Contractility.Phospho-Ser383-Elk-1 is localized to the mitotic spindles during cell cycle and interacts with mitotic kinase Aurora-A.The analysis of serum response factor expression in bone and soft tissue prostate cancer metastases.Control of cell death and mitochondrial fission by ERK1/2 MAP kinase signalling.PIASx acts as an Elk-1 coactivator by facilitating derepression.Elk-1 interacts with dynein upon serum stimulation but independent of serine 383 phosphorylation.Overlapping promoter targeting by Elk-1 and other divergent ETS-domain transcription factor family membersRegulation of ERK1/2 phosphorylation by acute and chronic morphine - implications for the role of cAMP-responsive element binding factor (CREB)-dependent and Ets-like protein-1 (Elk-1)-dependent transcription; small interfering RNA-based strategy.Translation inhibition during cell cycle arrest and apoptosis: Mcl-1 is a novel target for RNA binding protein CUGBP2.Elk1 and SRF transcription factors convey basal transcription and mediate glucose response via their binding sites in the human LXRB gene promoter.The ETS domain transcription factor Elk-1 regulates the expression of its partner protein, SRF.Identification and expression of SRF targeted by miR-133a during early development of Paralichthys olivaceus.Transcriptional Regulation of EGR1 by EGF and the ERK Signaling Pathway in Prostate Cancer Cells.Serum response factor modulates neuron survival during peripheral axon injury.Opposing effects of Elk-1 multisite phosphorylation shape its response to ERK activation
P2860
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P2860
Ternary complex factor-serum response factor complex-regulated gene activity is required for cellular proliferation and inhibition of apoptotic cell death.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Ternary complex factor-serum r ...... ition of apoptotic cell death.
@en
type
label
Ternary complex factor-serum r ...... ition of apoptotic cell death.
@en
prefLabel
Ternary complex factor-serum r ...... ition of apoptotic cell death.
@en
P2093
P2860
P1476
Ternary complex factor-serum r ...... ition of apoptotic cell death.
@en
P2093
Amanda O'donnell
Andrew D Sharrocks
Andy Hayes
Anne Seifert
Elaine R Vickers
Leo A H Zeef
P2860
P304
10340-10351
P356
10.1128/MCB.24.23.10340-10351.2004
P407
P577
2004-12-01T00:00:00Z