The ubiquitous transcription factor Oct-1 and the liver-specific factor HNF-1 are both required to activate transcription of a hepatitis B virus promoter.
about
PBX2 and PBX3, new homeobox genes with extensive homology to the human proto-oncogene PBX1Krüppel-like factor 15 activates hepatitis B virus gene expression and replicationIdentification of interacting transcription factors regulating tissue gene expression in humanMechanism of suppression of hepatitis B virus precore RNA transcription by a frequent double mutation.Transcriptional repression of human hepatitis B virus genes by a bZIP family member, E4BP4.Nuclear covalently closed circular viral genomic DNA in the liver of hepatocyte nuclear factor 1 alpha-null hepatitis B virus transgenic miceTransgenic expression of entire hepatitis B virus in mice induces hepatocarcinogenesis independent of chronic liver injuryEnhancer I predominance in hepatitis B virus gene expression.Transcriptional and posttranscriptional control of hepatitis B virus gene expressionA compilation of composite regulatory elements affecting gene transcription in vertebratesMembers of the nuclear receptor superfamily regulate transcription from the hepatitis B virus nucleocapsid promoter.Oct-1 acts as a transcriptional repressor on the C-reactive protein promoterHepatocyte nuclear factor 1α downregulates HBV gene expression and replication by activating the NF-κB signaling pathwayRepression of enhancer II activity by a negative regulatory element in the hepatitis B virus genome.Transcriptional regulation of precore and pregenomic RNAs of hepatitis B virus.Both muscle-specific and ubiquitous nuclear factors are required for muscle-specific expression of the myosin heavy-chain beta gene in cultured cellsPromoter-specific transactivation of hepatitis B virus transcription by a glutamine- and proline-rich domain of hepatocyte nuclear factor 1.Hepatocyte nuclear factor 1 and C/EBP are essential for the activity of the human apolipoprotein B gene second-intron enhancerA ubiquitous factor (HF-1a) and a distinct muscle factor (HF-1b/MEF-2) form an E-box-independent pathway for cardiac muscle gene expression.Control of hepatitis B virus at the level of transcription.The cooperative interaction between two motifs of an enhancer element of the chicken alpha A-crystallin gene, alpha CE1 and alpha CE2, confers lens-specific expression.Virus-host interplay in hepatitis B virus infection and epigenetic treatment strategies.Transactivation of the hepatitis B virus core promoter by the nuclear receptor FXRalpha.Conformational alteration of Oct-1 upon DNA binding dictates selectivity in differential interactions with related transcriptional coactivators.Dysregulated surface gene expression from disrupted hepatitis B virus genomes.Adaptively inferring human transcriptional subnetworks.A combination of upstream and proximal elements is required for efficient expression of the mouse renin promoter in cultured cells.The upstream region of the Rpe65 gene confers retinal pigment epithelium-specific expression in vivo and in vitro and contains critical octamer and E-box binding sites.T-13910 DNA variant associated with lactase persistence interacts with Oct-1 and stimulates lactase promoter activity in vitro.
P2860
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P2860
The ubiquitous transcription factor Oct-1 and the liver-specific factor HNF-1 are both required to activate transcription of a hepatitis B virus promoter.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
1991年學術文章
@zh
1991年學術文章
@zh-hant
name
The ubiquitous transcription f ...... a hepatitis B virus promoter.
@en
type
label
The ubiquitous transcription f ...... a hepatitis B virus promoter.
@en
prefLabel
The ubiquitous transcription f ...... a hepatitis B virus promoter.
@en
P2860
P356
P1476
The ubiquitous transcription f ...... a hepatitis B virus promoter.
@en
P2860
P304
P356
10.1128/MCB.11.3.1353
P407
P577
1991-03-01T00:00:00Z