Inactivation of menin, the product of the multiple endocrine neoplasia type 1 gene, inhibits the commitment of multipotential mesenchymal stem cells into the osteoblast lineage.
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Menin is required for bone morphogenetic protein 2- and transforming growth factor beta-regulated osteoblastic differentiation through interaction with Smads and Runx2Menin suppresses osteoblast differentiation by antagonizing the AP-1 factor, JunDGrowth hormone-secreting tumors: genetic aspects and data from animal modelsLoss of menin mediated by endothelial cells treated with CoPP is associated with increased maturation of adipocytesParathyroid hormone-responsive Smad3-related factor, Tmem119, promotes osteoblast differentiation and interacts with the bone morphogenetic protein-Runx2 pathwayZinc-modified Calcium Silicate Coatings Promote Osteogenic Differentiation through TGF-β/Smad Pathway and Osseointegration in Osteopenic RabbitsMenin is a regulator of the stress response in Drosophila melanogasterOsteoblast menin regulates bone mass in vivo.The Tissue Fibrinolytic System Contributes to the Induction of Macrophage Function and CCL3 during Bone Repair in MiceTomorrow's skeleton staff: mesenchymal stem cells and the repair of bone and cartilage.Consequence of Menin Deficiency in Mouse Adipocytes Derived by In Vitro DifferentiationPost-surgical follow-up of primary hyperparathyroidism associated with multiple endocrine neoplasia type 1.Menin induces endodermal differentiation in aggregated P19 stem cells by modulating the retinoic acid receptors.Loss of menin in osteoblast lineage affects osteocyte-osteoclast crosstalk causing osteoporosisTumor Suppressors and Endodermal Differentiation of P19 Embryonic Stem Cells.Menin and bone metabolism.TGF-β/BMP signaling pathway is involved in cerium-promoted osteogenic differentiation of mesenchymal stem cells.Twenty years of menin: emerging opportunities for restoration of transcription in MEN1.Tissue-type plasminogen activator deficiency delays bone repair: roles of osteoblastic proliferation and vascular endothelial growth factor.A novel regulatory role for stromal-derived factor-1 signaling in bone morphogenic protein-2 osteogenic differentiation of mesenchymal C2C12 cells.Nmp4/CIZ inhibits mechanically induced beta-catenin signaling activity in osteoblasts.The multiple endocrine neoplasia type 1 (MEN1) tumor suppressor regulates peroxisome proliferator-activated receptor gamma-dependent adipocyte differentiation.Lack of augmentation of tumor spectrum or severity in dual heterozygous Men1 and Rb1 knockout mice.Poly(l-Lactic Acid)/Gelatin Fibrous Scaffold Loaded with Simvastatin/Beta-Cyclodextrin-Modified Hydroxyapatite Inclusion Complex for Bone Tissue Regeneration.
P2860
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P2860
Inactivation of menin, the product of the multiple endocrine neoplasia type 1 gene, inhibits the commitment of multipotential mesenchymal stem cells into the osteoblast lineage.
description
2003 nî lūn-bûn
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2003年の論文
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2003年学术文章
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2003年学术文章
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2003年学术文章
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name
Inactivation of menin, the pro ...... s into the osteoblast lineage.
@en
type
label
Inactivation of menin, the pro ...... s into the osteoblast lineage.
@en
prefLabel
Inactivation of menin, the pro ...... s into the osteoblast lineage.
@en
P2093
P2860
P356
P1476
Inactivation of menin, the pro ...... s into the osteoblast lineage.
@en
P2093
Geoffrey N Hendy
Hideaki Sowa
Hiroshi Kaji
Kazuo Chihara
Kohei Miyazono
Lucie Canaff
Tatsuo Tsukamoto
Toru Yamaguchi
Toshitsugu Sugimoto
P2860
P304
21058-21069
P356
10.1074/JBC.M302044200
P407
P577
2003-03-20T00:00:00Z