JunD stabilization results in inhibition of normal intestinal epithelial cell growth through P21 after polyamine depletion.
about
RNA-binding proteins and microRNAs in gastrointestinal epithelial homeostasis and diseasesmiR-29b represses intestinal mucosal growth by inhibiting translation of cyclin-dependent kinase 2Polyamines regulate c-Myc translation through Chk2-dependent HuR phosphorylationInduced ATF-2 represses CDK4 transcription through dimerization with JunD inhibiting intestinal epithelial cell growth after polyamine depletionPolyamines regulate the stability of JunD mRNA by modulating the competitive binding of its 3' untranslated region to HuR and AUF1Chk2-dependent HuR phosphorylation regulates occludin mRNA translation and epithelial barrier functionPolyamine depletion induces nucleophosmin modulating stability and transcriptional activity of p53 in intestinal epithelial cellsMolecular characterization of mouse gastric zymogenic cells.Jnk2 deletion disrupts intestinal mucosal homeostasis and maturation by differentially modulating RNA-binding proteins HuR and CUGBP1.Polyamines and Gut Mucosal Homeostasis.Post-transcriptional regulation of MEK-1 by polyamines through the RNA-binding protein HuR modulating intestinal epithelial apoptosis.Modulation by miR-29b of intestinal epithelium homoeostasis through the repression of menin translation.Regulation of cyclin-dependent kinase 4 translation through CUG-binding protein 1 and microRNA-222 by polyaminesJunD enhances miR-29b levels transcriptionally and posttranscriptionally to inhibit proliferation of intestinal epithelial cells.Identification of mRNAs associated with alphaCP2-containing RNP complexesPolyamines regulate the stability of activating transcription factor-2 mRNA through RNA-binding protein HuR in intestinal epithelial cellsPolyamines regulate intestinal epithelial restitution through TRPC1-mediated Ca²+ signaling by differentially modulating STIM1 and STIM2.Polyamines and apoptosis.Polyamines regulate expression of E-cadherin and play an important role in control of intestinal epithelial barrier function.JunD represses transcription and translation of the tight junction protein zona occludens-1 modulating intestinal epithelial barrier function.Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous SpermidineRac1 promotes intestinal epithelial restitution by increasing Ca2+ influx through interaction with phospholipase C-(gamma)1 after wounding.Multiple facets of junD gene expression are atypical among AP-1 family members.Stabilization of XIAP mRNA through the RNA binding protein HuR regulated by cellular polyamines.Oxidative stress and oxidative damage in carcinogenesis.Induced JunD in intestinal epithelial cells represses CDK4 transcription through its proximal promoter region following polyamine depletion.Polyamine-modulated expression of c-myc plays a critical role in stimulation of normal intestinal epithelial cell proliferation.Transcriptional regulation of importin-α1 by JunD modulates subcellular localization of RNA-binding protein HuR in intestinal epithelial cells.Difluoromethylornithine in cancer: new advances.Differentiated intestinal epithelial cells express high levels of TGF-β receptors and exhibit increased sensitivity to growth inhibition.p53-dependent NDRG1 expression induces inhibition of intestinal epithelial cell proliferation but not apoptosis after polyamine depletion.Polyamine-modulated c-Myc expression in normal intestinal epithelial cells regulates p21Cip1 transcription through a proximal promoter region.Induced TRPC1 expression sensitizes intestinal epithelial cells to apoptosis by inhibiting NF-kappaB activation through Ca2+ influx.Effects of Si-Jun-Zi decoction polysaccharides on cell migration and gene expression in wounded rat intestinal epithelial cells.NF-kappaB-mediated IAP expression induces resistance of intestinal epithelial cells to apoptosis after polyamine depletion.Akt kinase activation blocks apoptosis in intestinal epithelial cells by inhibiting caspase-3 after polyamine depletion.Polyamine pathway inhibition as a novel therapeutic approach to treating neuroblastoma.RhoA stimulates IEC-6 cell proliferation by increasing polyamine-dependent Cdk2 activity.Activation of TGF-beta-Smad signaling pathway following polyamine depletion in intestinal epithelial cells.Polyamines are required for expression of Toll-like receptor 2 modulating intestinal epithelial barrier integrity.
P2860
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P2860
JunD stabilization results in inhibition of normal intestinal epithelial cell growth through P21 after polyamine depletion.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh
2002年學術文章
@zh-hant
name
JunD stabilization results in ...... P21 after polyamine depletion.
@en
type
label
JunD stabilization results in ...... P21 after polyamine depletion.
@en
prefLabel
JunD stabilization results in ...... P21 after polyamine depletion.
@en
P2093
P356
P1433
P1476
JunD stabilization results in ...... P21 after polyamine depletion.
@en
P2093
Ali Esmaili
Barbara L Bass
Eric D Strauch
Jaladanki N Rao
Jian-Ying Wang
P304
P356
10.1053/GAST.2002.35386
P407
P577
2002-09-01T00:00:00Z