Mouse models of hereditary hemorrhagic telangiectasia: recent advances and future challenges.
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Integrity of Narrow Epithelial Tubes in the C. elegans Excretory System Requires a Transient Luminal MatrixThe pathobiology of vascular malformations: insights from human and model organism genetics.Mice Lacking Endoglin in Macrophages Show an Impaired Immune Response.BMP-SMAD signalling output is highly regionalized in cardiovascular and lymphatic endothelial networks.Endoglin is required in Pax3-derived cells for embryonic blood vessel formation.Defective fluid shear stress mechanotransduction mediates hereditary hemorrhagic telangiectasia.Conditional knockout of activin like kinase-1 (ALK-1) leads to heart failure without maladaptive remodeling.Endoglin controls blood vessel diameter through endothelial cell shape changes in response to haemodynamic cues.The role of endoglin in post-ischemic revascularization.A mouse model of hereditary hemorrhagic telangiectasia generated by transmammary-delivered immunoblocking of BMP9 and BMP10.Endoglin prevents vascular malformation by regulating flow-induced cell migration and specification through VEGFR2 signalling.Experimental Animal Models of Arteriovenous Malformation: A Review.Endothelial cells respond to the direction of mechanical stimuli through SMAD signaling to regulate coronary artery size.An Ichor-dependent apical extracellular matrix regulates seamless tube shape and integrity.A novel association between high red blood cell alloimmunization rates and hereditary hemorrhagic telangiectasia.Reduced activin receptor-like kinase 1 activity promotes cardiac fibrosis in heart failure.Endoglin and alk1 as therapeutic targets for hereditary hemorrhagic telangiectasia.Endoglin moves and shapes endothelial cells.Bone Morphogenetic Protein 9 Reduces Cardiac Fibrosis and Improves Cardiac Function in Heart Failure.Hereditary hemorrhagic telangiectasia: diagnosis and management from the hematologist's perspectiveSoluble endoglin regulates expression of angiogenesis-related proteins and induction of arteriovenous malformations in a mouse model of hereditary hemorrhagic telangiectasia
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P2860
Mouse models of hereditary hemorrhagic telangiectasia: recent advances and future challenges.
description
2015 nî lūn-bûn
@nan
2015年の論文
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2015年学术文章
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2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
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2015年學術文章
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name
Mouse models of hereditary hem ...... dvances and future challenges.
@en
type
label
Mouse models of hereditary hem ...... dvances and future challenges.
@en
prefLabel
Mouse models of hereditary hem ...... dvances and future challenges.
@en
P2860
P356
P1476
Mouse models of hereditary hem ...... dvances and future challenges.
@en
P2093
P2860
P356
10.3389/FGENE.2015.00025
P577
2015-02-18T00:00:00Z