Pathogenic CD8+ T cells in experimental cerebral malaria.
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Perivascular Arrest of CD8+ T Cells Is a Signature of Experimental Cerebral MalariaMolecular basis of human cerebral malaria developmentInhibiting the Mammalian target of rapamycin blocks the development of experimental cerebral malaria.Targeting glutamine metabolism rescues mice from late-stage cerebral malaria.Oxidative insult can induce malaria-protective trait of sickle and fetal erythrocytes.The Deubiquitinating Enzyme Cylindromatosis Dampens CD8+ T Cell Responses and Is a Critical Factor for Experimental Cerebral Malaria and Blood-Brain Barrier DamageTargeting Angiotensin II Type-1 Receptor (AT1R) Inhibits the Harmful Phenotype of Plasmodium-Specific CD8+ T Cells during Blood-Stage Malaria.miR-155 Modifies Inflammation, Endothelial Activation and Blood-Brain Barrier Dysfunction in Cerebral Malaria.Investigating proteasome inhibitors as potential adjunct therapies for experimental cerebral malaria.Gamma Interferon Mediates Experimental Cerebral Malaria by Signaling within Both the Hematopoietic and Nonhematopoietic Compartments.Cytokines and Chemokines in Cerebral Malaria Pathogenesis.Small molecule-based inhibition of MEK1/2 proteins dampens inflammatory responses to malaria, reduces parasite load, and mitigates pathogenic outcomes.Myeloid expression of the AP-1 transcription factor JUNB modulates outcomes of type 1 and type 2 parasitic infections.Pathogenic CD8+ T Cells Cause Increased Levels of VEGF-A in Experimental Malaria-Associated Acute Respiratory Distress Syndrome, but Therapeutic VEGFR Inhibition Is Not Effective.Targeting the master regulator mTOR: a new approach to prevent the neurological of consequences of parasitic infections?Profiling MHC II immunopeptidome of blood-stage malaria reveals that cDC1 control the functionality of parasite-specific CD4 T cells.Doxycycline inhibits experimental cerebral malaria by reducing inflammatory immune reactions and tissue-degrading mediators.A malaria protein factor induces IL-4 production by dendritic cells via PI3K-Akt-NF-κB signaling independent of MyD88/TRIF and promotes Th2 response.Interleukin-15 Complex Treatment Protects Mice from Cerebral Malaria by Inducing Interleukin-10-Producing Natural Killer Cells.Common PIEZO1 Allele in African Populations Causes RBC Dehydration and Attenuates Plasmodium Infection.Inhibition of hypoxia-associated response and kynurenine production in response to hyperbaric oxygen as mechanisms involved in protection against experimental cerebral malaria.Transcriptomic Studies of Malaria: a Paradigm for Investigation of Systemic Host-Pathogen InteractionsTargeting the IL33-NLRP3 axis improves therapy for experimental cerebral malariaA Cross-Stage Antigen Contributes to the Development of Experimental Cerebral Malaria
P2860
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P2860
Pathogenic CD8+ T cells in experimental cerebral malaria.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Pathogenic CD8+ T cells in experimental cerebral malaria.
@en
type
label
Pathogenic CD8+ T cells in experimental cerebral malaria.
@en
prefLabel
Pathogenic CD8+ T cells in experimental cerebral malaria.
@en
P2860
P50
P1476
Pathogenic CD8+ T cells in experimental cerebral malaria.
@en
P2093
Chek Meng Poh
Shanshan Wu Howland
P2860
P2888
P304
P356
10.1007/S00281-015-0476-6
P577
2015-03-13T00:00:00Z
P5875
P6179
1027257517