Revisiting the NaCl cotransporter regulation by with-no-lysine kinases. - Wikidata - awgldk - TriplyDB

Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

http://www.wikidata.org/entity/Q38383563

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Degradation by Cullin 3 and effect on WNK kinases suggest a role of KLHL2 in the pathogenesis of Familial Hyperkalemic Hypertension Glucocorticoids Induce Nondipping Blood Pressure by Activating the Thiazide-Sensitive Cotransporter.Physiological role of SLC12 family members in the kidney.Basolateral Kir4.1 activity in the distal convoluted tubule regulates K secretion by determining NaCl cotransporter activity Potassium Homeostasis: The Knowns, the Unknowns, and the Health Benefits.Unique chloride-sensing properties of WNK4 permit the distal nephron to modulate potassium homeostasis.Potassium Supplementation Prevents Sodium Chloride Cotransporter Stimulation During Angiotensin II Hypertension.Potassium Sensing by Renal Distal Tubules Requires Kir4.1.Functional assessment of sodium chloride cotransporter NCC mutants in polarized mammalian epithelial cells.In Primary Aldosteronism, Mineralocorticoids Influence Exosomal Sodium-Chloride Cotransporter Abundance.Pleiotropic Actions of FGF23.Role of the alternative splice variant of NCC in blood pressure control

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Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

http://www.wikidata.org/entity/Q38383563

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

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2015年论文

@zh-cn

name

Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

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type

label

Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

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prefLabel

Revisiting the NaCl cotransporter regulation by with-no-lysine kinases.

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AJPCELL.00065.2015

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American Journal of Physiology - Cell Physiology

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Revisiting the NaCl cotransporter regulation by with-no-lysine kinases

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Silvana Bazúa-Valenti

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P2860

WNK3 kinase is a positive regulator of NKCC2 and NCC, renal cation-Cl- cotransporters required for normal blood pressure homeostasis

The thiazide-sensitive Na-Cl cotransporter is regulated by a WNK kinase signaling complex

Similar effects of all WNK3 variants on SLC12 cotransporters

KLHL3 mutations cause familial hyperkalemic hypertension by impairing ion transport in the distal nephron

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

WNK2 kinase is a novel regulator of essential neuronal cation-chloride cotransporters

The WNK-regulated SPAK/OSR1 kinases directly phosphorylate and inhibit the K+-Cl- co-transporters

Impaired KLHL3-mediated ubiquitination of WNK4 causes human hypertension

Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4

Molecular pathogenesis of inherited hypertension with hyperkalemia: the Na-Cl cotransporter is inhibited by wild-type but not mutant WNK4

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C779-91

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scholarly article

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10.1152/AJPCELL.00065.2015

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