Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Decreased epithelial progesterone receptor A at the window of receptivity is required for preparation of the endometrium for embryo attachment.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Chao Dong

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Linghong Wang

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Xiulan Su

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Ya-Guang Xi

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P2860

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

Impaired KLHL3-mediated ubiquitination of WNK4 causes human hypertension

Molecular pathogenesis of inherited hypertension with hyperkalemia: the Na-Cl cotransporter is inhibited by wild-type but not mutant WNK4

The thiazide-sensitive Na+-Cl- cotransporter: molecular biology, functional properties, and regulation by WNKs

Rare independent mutations in renal salt handling genes contribute to blood pressure variation

WNK3 and WNK4 amino-terminal domain defines their effect on the renal Na+-Cl- cotransporter

STK39 polymorphism is associated with essential hypertension: a systematic review and meta-analysis

Using haplotype blocks to map human complex trait loci

Hyperplasia of the juxtaglomerular complex with hyperaldosteronism and hypokalemic alkalosis. A new syndrome

Functional expression of mutations in the human NaCl cotransporter: evidence for impaired routing mechanisms in Gitelman's syndrome

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10.1093/ABBS/GMV020

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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Thiazide-sensitive Na+-Cl- cotransporter: genetic polymorphisms and human diseases.

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