K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study. - Wikidata - awgldk - TriplyDB

K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

http://www.wikidata.org/entity/Q38452428

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Mutations in APC, CTNNB1 and K-ras genes and expression of hMLH1 in sporadic colorectal carcinomas from the Netherlands Cohort Study.PHLPP is a negative regulator of RAF1, which reduces colorectal cancer cell motility and prevents tumor progression in mice Cell carriage, delivery, and selective replication of an oncolytic virus in tumor in patients Molecular pathological epidemiology of colorectal neoplasia: an emerging transdisciplinary and interdisciplinary field MicroRNAs as growth regulators, their function and biomarker status in colorectal cancer Effectiveness and safety of monoclonal antibodies for metastatic colorectal cancer treatment: systematic review and meta-analysis KRAS mutation in stage III colon cancer and clinical outcome following intergroup trial CALGB 89803 BRAF vs RAS oncogenes: are mutations of the same pathway equal? Differential signalling and therapeutic implications Novel KRAS gene mutations in sporadic colorectal cancer Mutated KRAS results in overexpression of DUSP4, a MAP-kinase phosphatase, and SMYD3, a histone methyltransferase, in rectal carcinomas.Early life exposure to famine and colorectal cancer risk: a role for epigenetic mechanisms.Phase I pharmacokinetic and pharmacodynamic study of cetuximab, irinotecan and sorafenib in advanced colorectal cancer Experiences from treatment-predictive KRAS testing; high mutation frequency in rectal cancers from females and concurrent mutations in the same tumor.Targeted KRAS mutation assessment on patient tumor histologic material in real time diagnostics.Dietary, lifestyle and clinicopathological factors associated with BRAF and K-ras mutations arising in distinct subsets of colorectal cancers in the EPIC Norfolk study Oncogenic ras-induced down-regulation of autophagy mediator Beclin-1 is required for malignant transformation of intestinal epithelial cells Cetuximab plus oxaliplatin/leucovorin/5-fluorouracil in first-line metastatic gastric cancer: a phase II study of the Arbeitsgemeinschaft Internistische Onkologie (AIO).Characterization of rare transforming KRAS mutations in sporadic colorectal cancer.MGMT promoter hypermethylation and K-RAS, PTEN and TP53 mutations in tamoxifen-exposed and non-exposed endometrial cancer cases Body size, physical activity and risk of colorectal cancer with or without the CpG island methylator phenotype (CIMP).NCI60 cancer cell line panel data and RNAi analysis help identify EAF2 as a modulator of simvastatin and lovastatin response in HCT-116 cells.Pharmacogenomics: Current State-of-the-Art A flexible approach for highly multiplexed candidate gene targeted resequencing.The activating mutation R201C in GNAS promotes intestinal tumourigenesis in Apc(Min/+) mice through activation of Wnt and ERK1/2 MAPK pathways.The involvement of Kras gene 3'-UTR polymorphisms in risk of cancer and influence on patient response to anti-EGFR therapy in metastatic colorectal cancer: a meta-analysis.Biological and molecular effects of small molecule kinase inhibitors on low-passage human colorectal cancer cell lines.Computational analysis of KRAS mutations: implications for different effects on the KRAS p.G12D and p.G13D mutations.Association between specific KRAS mutations and the clinicopathological characteristics of colorectal tumors.The role of oncogenes in gastrointestinal cancer.EGFR Signaling in Colorectal Carcinoma.Colorectal carcinoma: Pathologic aspects Kras gene mutation and RASSF1A, FHIT and MGMT gene promoter hypermethylation: indicators of tumor staging and metastasis in adenocarcinomatous sporadic colorectal cancer in Indian population IMP-1 displays cross-talk with K-Ras and modulates colon cancer cell survival through the novel proapoptotic protein CYFIP2 Context-dependent interpretation of the prognostic value of BRAF and KRAS mutations in colorectal cancer.Frequent mutations of KRAS in addition to BRAF in colorectal serrated adenocarcinoma.Identification of the angiogenic gene signature induced by EGF and hypoxia in colorectal cancer.A mill based instrument and software system for dissecting slide-mounted tissue that provides digital guidance and documentation.KRAS and BRAF gene mutations and DNA mismatch repair status in Chinese colorectal carcinoma patients KRAS, NRAS, PIK3CA exon 20, and BRAF genotypes in synchronous and metachronous primary colorectal cancers diagnostic and therapeutic implications Identification of candidate driver genes in common focal chromosomal aberrations of microsatellite stable colorectal cancer

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K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

http://www.wikidata.org/entity/Q38452428

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K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

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K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

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K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

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K-ras oncogene mutations in sporadic colorectal cancer in The Netherlands Cohort Study.

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Adriaan P de Bruïne

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Anton F P M de Goeij

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Guido M J M Roemen

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Kim M Smits

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Marco M M Pachen

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Marjolein H F M Lentjes

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Matty P Weijenberg

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Mirian Brink

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R Alexandra Goldbohm

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703-710

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scholarly article

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10.1093/CARCIN/BGG009

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4

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24

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Piet A van den Brandt

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2003-04-01T00:00:00Z

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12727799

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colorectal cancer