FliT acts as an anti-FlhD2C2 factor in the transcriptional control of the flagellar regulon in Salmonella enterica serovar typhimurium.
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Regulatory principles governing Salmonella and Yersinia virulenceLessons in Fundamental Mechanisms and Diverse Adaptations from the 2015 Bacterial Locomotion and Signal Transduction MeetingA comprehensive genetic characterization of bacterial motilityStructural insight into the regulatory mechanisms of interactions of the flagellar type III chaperone FliT with its binding partnersStructural insight of a concentration-dependent mechanism by which YdiV inhibits Escherichia coli flagellum biogenesis and motilityEAL domain protein YdiV acts as an anti-FlhD4C2 factor responsible for nutritional control of the flagellar regulon in Salmonella enterica Serovar TyphimuriumProtein export according to schedule: architecture, assembly, and regulation of type III secretion systems from plant- and animal-pathogenic bacteria.Multiple promoters contribute to swarming and the coordination of transcription with flagellar assembly in Salmonella.Analysis of factors that affect FlgM-dependent type III secretion for protein purification with Salmonella enterica serovar Typhimurium.The role of the FliD C-terminal domain in pentamer formation and interaction with FliT.Genetic and molecular characterization of flagellar assembly in Shewanella oneidensis.The flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.A nutrient-tunable bistable switch controls motility in Salmonella enterica serovar Typhimurium.FliZ induces a kinetic switch in flagellar gene expression.FliT selectively enhances proteolysis of FlhC subunit in FlhD4C2 complex by an ATP-dependent protease, ClpXP.FlgM as a secretion moiety for the development of an inducible type III secretion systemYdiV: a dual function protein that targets FlhDC for ClpXP-dependent degradation by promoting release of DNA-bound FlhDC complex.FliZ is a global regulatory protein affecting the expression of flagellar and virulence genes in individual Xenorhabdus nematophila bacterial cells.The effect of cell growth phase on the regulatory cross-talk between flagellar and Spi1 virulence gene expression.Refining the binding of the Escherichia coli flagellar master regulator, FlhD4C2, on a base-specific levelC-ring requirement in flagellar type III secretion is bypassed by FlhDC upregulation.The transcript from the σ(28)-dependent promoter is translationally inert in the expression of the σ(28)-encoding gene fliA in the fliAZ operon of Salmonella enterica serovar TyphimuriumPerturbation of FliL interferes with Proteus mirabilis swarmer cell gene expression and differentiation.Flagellin Is Required for Host Cell Invasion and Normal Salmonella Pathogenicity Island 1 Expression by Salmonella enterica Serovar Paratyphi AControl of gene expression by type III secretory activity.FliZ Is a posttranslational activator of FlhD4C2-dependent flagellar gene expression.Flagellar biogenesis of Xanthomonas campestris requires the alternative sigma factors RpoN2 and FliA and is temporally regulated by FlhA, FlhB, and FlgM.Coordinating assembly of a bacterial macromolecular machine.Recognition and targeting mechanisms by chaperones in flagellum assembly and operationUpward mobility and alternative lifestyles: a report from the 10th biennial meeting on Bacterial Locomotion and Signal TransductionFliZ acts as a repressor of the ydiV gene, which encodes an anti-FlhD4C2 factor of the flagellar regulon in Salmonella enterica serovar typhimurium.Mathematical model of flagella gene expression dynamics in Salmonella enterica serovar typhimurium.RflM mediates target specificity of the RcsCDB phosphorelay system for transcriptional repression of flagellar synthesis in Salmonella enterica.Genetic analysis of revertants isolated from the rod-fragile fliF mutant of Salmonella.RpoN2- and FliA-regulated fliTX is indispensible for flagellar motility and virulence in Xanthomonas oryzae pv. oryzae.FliS modulates FlgM activity by acting as a non-canonical chaperone to control late flagellar gene expression, motility and biofilm formation in Yersinia pseudotuberculosis.Purification, crystallization and preliminary X-ray analysis of FliT, a bacterial flagellar substrate-specific export chaperone.Defects in the flagellar motor increase synthesis of poly-γ-glutamate in Bacillus subtilis.RsmC of Erwinia carotovora subsp. carotovora negatively controls motility, extracellular protein production, and virulence by binding FlhD and modulating transcriptional activity of the master regulator, FlhDC.Mutually repressing repressor functions and multi-layered cellular heterogeneity regulate the bistable Salmonella fliC census.
P2860
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P2860
FliT acts as an anti-FlhD2C2 factor in the transcriptional control of the flagellar regulon in Salmonella enterica serovar typhimurium.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
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2006年论文
@zh-cn
name
FliT acts as an anti-FlhD2C2 f ...... enterica serovar typhimurium.
@en
type
label
FliT acts as an anti-FlhD2C2 f ...... enterica serovar typhimurium.
@en
prefLabel
FliT acts as an anti-FlhD2C2 f ...... enterica serovar typhimurium.
@en
P2860
P356
P1476
FliT acts as an anti-FlhD2C2 f ...... enterica serovar typhimurium.
@en
P2093
Kazuhiro Kutsukake
Shouji Yamamoto
P2860
P304
P356
10.1128/JB.00799-06
P407
P577
2006-09-01T00:00:00Z