Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation.
about
Oxidative stress and cerebral endothelial cells: regulation of the blood-brain-barrier and antioxidant based interventionsAmyloid-β and proinflammatory cytokines utilize a prion protein-dependent pathway to activate NADPH oxidase and induce cofilin-actin rods in hippocampal neuronsSepsis-induced selective parvalbumin interneuron phenotype loss and cognitive impairments may be mediated by NADPH oxidase 2 activation in mice.The developing utility of zebrafish models for cognitive enhancers researchTranslational research in immune senescence: assessing the relevance of current models.New insights on NOX enzymes in the central nervous system.Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse modelsOxidative stress, redox signalling and endothelial dysfunction in ageing-related neurodegenerative diseases: a role of NADPH oxidase 2.Increased fragmentation of sleep-wake cycles in the 5XFAD mouse model of Alzheimer's diseaseProlonged diet induced obesity has minimal effects towards brain pathology in mouse model of cerebral amyloid angiopathy: implications for studying obesity-brain interactions in mice.Brain injury caused by HIV protease inhibitors: role of lipodystrophy and insulin resistance.The 1027th target candidate in stroke: Will NADPH oxidase hold up?NOX2 deficiency attenuates markers of adiposopathy and brain injury induced by high-fat diet.Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's diseaseDietary and donepezil modulation of mTOR signaling and neuroinflammation in the brain.NADPH oxidase in brain injury and neurodegenerative disordersNADPH oxidase and neurodegenerationMyeloid-specific deletion of NOX2 prevents the metabolic and neurologic consequences of high fat diet.Innate immunity in Alzheimer's disease: the relevance of animal models?Minocycline reduces spontaneous hemorrhage in mouse models of cerebral amyloid angiopathy.Early stage drug treatment that normalizes proinflammatory cytokine production attenuates synaptic dysfunction in a mouse model that exhibits age-dependent progression of Alzheimer's disease-related pathology.A Mini-Review of the NADPH oxidases in Vascular Dementia: Correlation with NOXs and Risk Factors for VaD.Cilostazol Suppresses Aβ-induced Neurotoxicity in SH-SY5Y Cells through Inhibition of Oxidative Stress and MAPK Signaling Pathway.Effects and Mechanism of Huannao Yicong Decoction Extract on the Ethology of Transgenic APP/PS1 Mice.
P2860
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P2860
Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation.
@en
type
label
Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation.
@en
prefLabel
Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation.
@en
P2093
P2860
P1476
Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation
@en
P2093
Alecia G Knight
Annadora J Bruce-Keller
Daret St Clair
Jessica M McMullen
Linda J Van Eldik
M Paul Murphy
Paulina R Davis
Sunita Gupta
Tina L Beckett
P2860
P304
P356
10.1016/J.NBD.2011.07.012
P50
P577
2011-07-28T00:00:00Z