Ras triggers acidosis-induced activation of the extracellular-signal-regulated kinase pathway in cardiac myocytes.
about
The Na(+) /H(+) exchanger and pH regulation in the heartp90 activation contributes to cerebral ischemic damage via phosphorylation of Na+/H+ exchanger isoform 1.Ca(2+)/calmodulin-dependent protein kinase II contributes to intracellular pH recovery from acidosis via Na(+)/H(+) exchanger activation.B-Raf associates with and activates the NHE1 isoform of the Na+/H+ exchanger.Sustained Intracellular Acidosis Triggers the Na⁺/H⁺ Exchager-1 Activation in Glutamate Excitotoxicity.The reduced myofilament responsiveness to calcium contributes to the negative force-frequency relationship in rat cardiomyocytes: role of reactive oxygen species and p-38 map kinase.Cycloprodigiosin hydrochloride activates the Ras-PI3K-Akt pathway and suppresses protein synthesis inhibition-induced apoptosis in PC12 cells.Involment of RAS/ERK1/2 signaling and MEF2C in miR-155-3p inhibition-triggered cardiomyocyte differentiation of embryonic stem cell.Exercise alters the regulation of myocardial Na(+)/H(+) exchanger-1 activity.
P2860
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P2860
Ras triggers acidosis-induced activation of the extracellular-signal-regulated kinase pathway in cardiac myocytes.
description
2006 nî lūn-bûn
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name
Ras triggers acidosis-induced ...... e pathway in cardiac myocytes.
@en
type
label
Ras triggers acidosis-induced ...... e pathway in cardiac myocytes.
@en
prefLabel
Ras triggers acidosis-induced ...... e pathway in cardiac myocytes.
@en
P2093
P2860
P356
P1433
P1476
Ras triggers acidosis-induced ...... e pathway in cardiac myocytes.
@en
P2093
Metin Avkiran
Robert S Haworth
Semjidmaa Dashnyam
P2860
P304
P356
10.1042/BJ20051628
P407
P577
2006-11-01T00:00:00Z