Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
about
Alternative splicing: the pledge, the turn, and the prestige : The key role of alternative splicing in human biological systems.Intron retention as a component of regulated gene expression programs.Transcriptome analysis of hypoxic cancer cells uncovers intron retention in EIF2B5 as a mechanism to inhibit translation.Intron retention in viruses and cellular genes: Detention, border controls and passports.Hypoxia leads to significant changes in alternative splicing and elevated expression of CLK splice factor kinases in PC3 prostate cancer cells.
P2860
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
description
2016 nî lūn-bûn
@nan
2016年の論文
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2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
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2016年论文
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2016年论文
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name
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
@en
type
label
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
@en
prefLabel
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
@en
P2093
P2860
P50
P1433
P1476
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.
@en
P2093
Christopher Sf Smowton
Keren Dawson
P2860
P2888
P356
10.1038/NPJGENMED.2016.20
P577
2016-07-20T00:00:00Z
P6179
1037815608