N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
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The ATP-grasp enzymesN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationMolecular identification of β-citrylglutamate hydrolase as glutamate carboxypeptidase 3NAAG peptidase inhibition in the periaqueductal gray and rostral ventromedial medulla reduces flinching in the formalin model of inflammationNAAG peptidase inhibitors block cognitive deficit induced by MK-801 and motor activation induced by d-amphetamine in animal models of schizophrenia.Advances in understanding the peptide neurotransmitter NAAG and appearance of a new member of the NAAG neuropeptide familyThe Concise Guide to PHARMACOLOGY 2013/14: G protein-coupled receptorsGlutamate carboxypeptidase II inhibition behaviorally and physiologically improves pyridoxine-induced neuropathy in rats.Selective CNS Uptake of the GCP-II Inhibitor 2-PMPA following Intranasal Administration.Glutamate carboxypeptidase II gene knockout attenuates oxidative stress and cortical apoptosis after traumatic brain injury.Effects of N-acetylaspartylglutamate (NAAG) peptidase inhibition on release of glutamate and dopamine in prefrontal cortex and nucleus accumbens in phencyclidine model of schizophreniaImmunohistological and electrophysiological evidence that N-acetylaspartylglutamate is a co-transmitter at the vertebrate neuromuscular junctionNAAG peptidase inhibitors and deletion of NAAG peptidase gene enhance memory in novel object recognition test.Presynaptic, release-regulating mGlu2 -preferring and mGlu3 -preferring autoreceptors in CNS: pharmacological profiles and functional roles in demyelinating diseaseBridging the gap between non-targeted stable isotope labeling and metabolic flux analysis.A role for the locus coeruleus in the analgesic efficacy of N-acetylaspartylglutamate peptidase (GCPII) inhibitors ZJ43 and 2-PMPA.NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer's, and Ethanol Intoxication.Mice lacking glutamate carboxypeptidase II develop normally, but are less susceptible to traumatic brain injury.Maternal inflammation leads to impaired glutamate homeostasis and up-regulation of glutamate carboxypeptidase II in activated microglia in the fetal/newborn rabbit brain.Vesicular uptake of N-acetylaspartylglutamate is catalysed by sialin (SLC17A5).N-acetylaspartylglutamate Inhibits Heroin Self-Administration and Heroin-Seeking Behaviors Induced by Cue or Priming in Rats.Enhanced Brain Delivery of 2-(Phosphonomethyl)pentanedioic Acid Following Intranasal Administration of Its γ-Substituted Ester Prodrugs.Blocking glutamate carboxypeptidase II inhibits glutamate excitotoxicity and regulates immune responses in experimental autoimmune encephalomyelitis.Group II Metabotropic Glutamate Receptors: Role in Pain Mechanisms and Pain Modulation
P2860
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P2860
N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
@en
type
label
N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
@en
prefLabel
N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
@en
P2860
P1476
N-acetylaspartylglutamate is an agonist at mGluR₃ in vivo and in vitro.
@en
P2093
Joseph H Neale
P2860
P304
P356
10.1111/J.1471-4159.2011.07380.X
P407
P577
2011-12-01T00:00:00Z