Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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P1476

Telomere uncapping at the crossroad between cell cycle arrest and carcinogenesis

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P2093

Camilla Trovesi

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Corinne Cassani

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Elisa Gobbini

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Maria Pia Longhese

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P2860

Shelterin-like proteins and Yku inhibit nucleolytic processing of Saccharomyces cerevisiae telomeres

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

Five dysfunctional telomeres predict onset of senescence in human cells

Extension of life-span by introduction of telomerase into normal human cells

Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes

Specific association of human telomerase activity with immortal cells and cancer

The DNA Damage Response: Making It Safe to Play with Knives

Sae2, Exo1 and Sgs1 collaborate in DNA double-strand break processing.

A mutant with a defect in telomere elongation leads to senescence in yeast.

The Saccharomyces telomere-binding protein Cdc13p interacts with both the catalytic subunit of DNA polymerase alpha and the telomerase-associated est1 protein

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e29901

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P31

scholarly article

P356

10.4161/MCO.29901

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P433

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P478

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P577

2014-07-28T00:00:00Z

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P5875

271104092

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P698

27308311

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P932

4905175

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