Is There a Role for Endogenous 5-HT in Gastrointestinal Motility? How Recent Studies Have Changed Our Understanding.

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Motility patterns in mouse colon: gastrointestinal dysfunction induced by anticancer chemotherapy.Prolonged intestinal transit and diarrhea in patients with an activating GUCY2C mutation.Exogenous serotonin regulates colorectal motility via the 5-HT2 and 5-HT3 receptors in the spinal cord of rats.

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Is There a Role for Endogenous 5-HT in Gastrointestinal Motility? How Recent Studies Have Changed Our Understanding.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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Pharmacological inhibition of gut-derived serotonin synthesis is a potential bone anabolic treatment for osteoporosis

Constitutive dimerization of human serotonin 5-HT4 receptors in living cells.

Alosetron.

Multiple mechanisms of fast excitatory synaptic transmission in the enteric nervous system.

Localized release of serotonin (5-hydroxytryptamine) by a fecal pellet regulates migrating motor complexes in murine colon.

The 5-HT3B subunit confers spontaneous channel opening and altered ligand properties of the 5-HT3 receptor.

Essential roles of enteric neuronal serotonin in gastrointestinal motility and the development/survival of enteric dopaminergic neurons.

By what mechanism does ondansetron inhibit colonic migrating motor complexes: does it require endogenous serotonin in the gut wall?

Mechanisms underlying distension-evoked peristalsis in guinea pig distal colon: is there a role for enterochromaffin cells?

Release of 5-hydroxytryptamine from the mucosa is not required for the generation or propagation of colonic migrating motor complexes.

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113-122

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scholarly article

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10.1007/978-3-319-27592-5_11

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English

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891

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Damien J Keating

Nick J. Spencer

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2016-01-01T00:00:00Z

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Is There a Role for Endogenous 5-HT in Gastrointestinal Motility? How Recent Studies Have Changed Our Understanding.

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P2860

Is There a Role for Endogenous 5-HT in Gastrointestinal Motility? How Recent Studies Have Changed Our Understanding.

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