Silencing of PINK1 inhibits insulin-like growth factor-1-mediated receptor activation and neuronal survival.

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Ubiquitin Ligases and Posttranslational Regulation of Energy in the Heart: The Hand that Feeds.Liver X Receptor Agonist GW3965 Regulates Synaptic Function upon Amyloid Beta Exposure in Hippocampal Neurons.

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Q39390991-4C605C36-1014-4739-B7BF-12A39C6FEBAE Q47235828-08A9F40D-8DC5-4166-98CE-E9F0CEFAADAC

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Silencing of PINK1 inhibits insulin-like growth factor-1-mediated receptor activation and neuronal survival.

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http://www.wikidata.org/entity/Q38926013

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2014 nî lūn-bûn

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2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

2014年论文

@zh

2014年论文

@zh-cn

name

Silencing of PINK1 inhibits in ...... ivation and neuronal survival.

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type

Item

label

Silencing of PINK1 inhibits in ...... ivation and neuronal survival.

@en

prefLabel

Silencing of PINK1 inhibits in ...... ivation and neuronal survival.

@en

P1476

Silencing of PINK1 inhibits in ...... tivation and neuronal survival

@en

P2093

Andrea Niño

http://www.w3.org/2001/XMLSchema#string

Gonzalo Arboleda

http://www.w3.org/2001/XMLSchema#string

Humberto Arboleda

http://www.w3.org/2001/XMLSchema#string

Liliana Rojas

http://www.w3.org/2001/XMLSchema#string

P2860

Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability

PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

Hexokinase activity is required for recruitment of parkin to depolarized mitochondria

A new cytosolic pathway from a Parkinson disease-associated kinase, BRPK/PINK1: activation of AKT via mTORC2

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

PINK1 is necessary for long term survival and mitochondrial function in human dopaminergic neurons

Feature Article: mTOR complex 2-Akt signaling at mitochondria-associated endoplasmic reticulum membranes (MAM) regulates mitochondrial physiology

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s12031-014-0479-0

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188-197

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scholarly article

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10.1007/S12031-014-0479-0

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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María J Contreras-Zárate

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2014-12-24T00:00:00Z

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269999106

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P6179

1039991791

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P698

25534921

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Silencing of PINK1 inhibits insulin-like growth factor-1-mediated receptor activation and neuronal survival.

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P2860

P2860

Silencing of PINK1 inhibits insulin-like growth factor-1-mediated receptor activation and neuronal survival.

description

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P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P5875

P6179

P698

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P5875

P6179

P698