SM protein Munc18-2 facilitates transition of Syntaxin 11-mediated lipid mixing to complete fusion for T-lymphocyte cytotoxicity.
about
Disrupted apical exocytosis of cargo vesicles causes enteropathy in FHL5 patients with Munc18-2 mutations.Syntaxin 4 mediates endosome recycling for lytic granule exocytosis in cytotoxic T-lymphocytes.Revisiting interaction specificity reveals neuronal and adipocyte Munc18 membrane fusion regulatory proteins differ in their binding interactions with partner SNARE Syntaxins.Two Disease-Causing SNAP-25B Mutations Selectively Impair SNARE C-terminal Assembly.Bi-Allelic Mutations in STXBP2 Reveal a Complementary Role for STXBP1 in Cytotoxic Lymphocyte Killing.
P2860
SM protein Munc18-2 facilitates transition of Syntaxin 11-mediated lipid mixing to complete fusion for T-lymphocyte cytotoxicity.
description
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2017年の論文
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name
SM protein Munc18-2 facilitate ...... for T-lymphocyte cytotoxicity.
@en
type
label
SM protein Munc18-2 facilitate ...... for T-lymphocyte cytotoxicity.
@en
prefLabel
SM protein Munc18-2 facilitate ...... for T-lymphocyte cytotoxicity.
@en
P2093
P2860
P356
P1476
SM protein Munc18-2 facilitate ...... for T-lymphocyte cytotoxicity.
@en
P2093
Margaret E McCormick
Maria L Sanmillan
Vineet V Kulkarni
Waldo A Spessott
P2860
P304
E2176-E2185
P356
10.1073/PNAS.1617981114
P407
P577
2017-03-06T00:00:00Z