Molecular Pathophysiology of Congenital Long QT Syndrome.
about
Sodium leak through K2P potassium channels and cardiac arrhythmia, an emerging themeContribution of two-pore K+ channels to cardiac ventricular action potential revealed using human iPSC-derived cardiomyocytes.SUMOylation determines the voltage required to activate cardiac IKs channels.Humans Vary, So Cardiac Models Should Account for That Too!Trigger vs. Substrate: Multi-Dimensional Modulation of QT-Prolongation Associated Arrhythmic Dynamics by a hERG Channel Activator.β-adrenergic stimulation augments transmural dispersion of repolarization via modulation of delayed rectifier currents IKs and IKr in the human ventricle.A hERG mutation E1039X produced a synergistic lesion on IKs together with KCNQ1-R174C mutation in a LQTS family with three compound mutations.Studying KCNQ1 Mutation and Drug Response in Type 1 Long QT Syndrome Using Patient-Specific Induced Pluripotent Stem Cell-Derived Cardiomyocytes.Development of a High-Throughput Flow Cytometry Assay to Monitor Defective Trafficking and Rescue of Long QT2 Mutant hERG Channels.Long QT Syndrome and Sinus Bradycardia-A Mini Review
P2860
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P2860
Molecular Pathophysiology of Congenital Long QT Syndrome.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Cooperativity of SV40 T antige ...... ormation of human fibroblasts.
@ast
Molecular Pathophysiology of Congenital Long QT Syndrome.
@en
type
label
Cooperativity of SV40 T antige ...... ormation of human fibroblasts.
@ast
Molecular Pathophysiology of Congenital Long QT Syndrome.
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prefLabel
Cooperativity of SV40 T antige ...... ormation of human fibroblasts.
@ast
Molecular Pathophysiology of Congenital Long QT Syndrome.
@en
P2093
P2860
P1476
Molecular Pathophysiology of Congenital Long QT Syndrome.
@en
P2093
C Terrenoire
K J Sampson
M S Bohnen
P2860
P304
P356
10.1152/PHYSREV.00008.2016
P577
2017-01-01T00:00:00Z