FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression. - Wikidata - awgldk - TriplyDB

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

http://www.wikidata.org/entity/Q39026147

about

FOXM1 induces a global methylation signature that mimics the cancer epigenome in head and neck squamous cell carcinoma FoxM1, a critical regulator of oxidative stress during oncogenesis Prognostic Value of FOXM1 in Patients with Malignant Solid Tumor: A Meta-Analysis and System Review FOXM1: an emerging master regulator of DNA damage response and genotoxic agent resistance Epithelial-mesenchymal transition and cancer stemness: the Twist1-Bmi1 connection GFAP-Cre-mediated transgenic activation of Bmi1 results in pituitary tumors Recent advances of highly selective CDK4/6 inhibitors in breast cancer Forkhead box M1 transcription factor: a novel target for cancer therapy.Prevention of senescence progression in reversibly immortalized human ensheathing glia permits their survival after deimmortalization.Increased FOXM1 expression can stimulate DNA repair in normal hepatocytes in vivo but also increases nuclear foci associated with senescence.Pro-proliferative FoxM1 is a target of p53-mediated repression.microRNA-141 regulates BMI1 expression and induces senescence in human diploid fibroblasts.Multiple faces of FoxM1 transcription factor: lessons from transgenic mouse models.Activating CAR and β-catenin induces uncontrolled liver growth and tumorigenesis.Estrogen receptor α-coupled Bmi1 regulation pathway in breast cancer and its clinical implications.Advances and developments in the use of human mesenchymal stem cells - a few considerations.Foxm1 mediates LIF/Stat3-dependent self-renewal in mouse embryonic stem cells and is essential for the generation of induced pluripotent stem cells Expression Analysis of Genes Involved in the RB/E2F Pathway in Astrocytic Tumors.Activation of FoxM1 Revitalizes the Replicative Potential of Aged β-Cells in Male Mice and Enhances Insulin Secretion.The requirement for cyclin D function in tumor maintenance.Forkhead box M1 is regulated by heat shock factor 1 and promotes glioma cells survival under heat shock stress A positive feedback loop regulates the expression of polycomb group protein BMI1 via WNT signaling pathway.Novel functions of FoxM1: from molecular mechanisms to cancer therapy PKCι counteracts oxidative stress by regulating Hsc70 in an esophageal cancer cell line.Heterozygous knockout of the Bmi-1 gene causes an early onset of phenotypes associated with brain aging.The roles of FOXM1 in pancreatic stem cells and carcinogenesis BMI1 as a novel target for drug discovery in cancer.FOX(M1) news--it is cancer.Polycomb group proteins and MYC: the cancer connection.Insights into a Critical Role of the FOXO3a-FOXM1 Axis in DNA Damage Response and Genotoxic Drug Resistance.Reactive oxygen species generation is essential for cisplatin-induced accelerated senescence in hepatocellular carcinoma.Silencing of FOXM1 transcription factor expression by adenovirus-mediated RNA interference inhibits human hepatocellular carcinoma growth.Roles of FoxM1 in cell regulation and breast cancer targeting therapy.Subnuclear distribution of SSX regulates its function.Adenovirus-mediated RNA interference targeting FOXM1 transcription factor suppresses cell proliferation and tumor growth of nasopharyngeal carcinoma.FoxM1 in tumorigenicity of the neuroblastoma cells and renewal of the neural progenitors.FOXM1 regulates proliferation, senescence and oxidative stress in keratinocytes and cancer cells Targeting FoxM1 effectively retards p53-null lymphoma and sarcoma.Fkh1 and Fkh2 associate with Sir2 to control CLB2 transcription under normal and oxidative stress conditions.Sp1 and c-Myc regulate transcription of BMI1 in nasopharyngeal carcinoma.

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P2860

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

http://www.wikidata.org/entity/Q39026147

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

2008年论文

@zh

2008年论文

@zh-cn

name

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@en

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@nl

type

label

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@en

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@nl

prefLabel

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@en

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression.

@nl

P1433

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P1433

Journal of Biological Chemistry

P1476

FoxM1c counteracts oxidative stress-induced senescence and stimulates Bmi-1 expression

@en

P2093

Alice M S Cheung

http://www.w3.org/2001/XMLSchema#string

David K Smith

http://www.w3.org/2001/XMLSchema#string

Goberdhan P Dimri

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Kwok-Ming Yao

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Samuel K M Li

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Wai Ying Leung

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P2860

The Forkhead box M1 protein regulates the transcription of the estrogen receptor alpha in breast cancer cells

Chk2 mediates stabilization of the FoxM1 transcription factor to stimulate expression of DNA repair genes

Hepatocyte nuclear factor 3/fork head homolog 11 is expressed in proliferating epithelial and mesenchymal cells of embryonic and adult tissues

Molecular analysis of a novel winged helix protein, WIN. Expression pattern, DNA binding property, and alternative splicing within the DNA binding domain

Increased levels of the FoxM1 transcription factor accelerate development and progression of prostate carcinomas in both TRAMP and LADY transgenic mice

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Foxm1b transcription factor is essential for development of hepatocellular carcinomas and is negatively regulated by the p19ARF tumor suppressor

Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation

The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells

Mel-18, a polycomb group protein, regulates cell proliferation and senescence via transcriptional repression of Bmi-1 and c-Myc oncoproteins

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16545-16553

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scholarly article

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10.1074/JBC.M709604200

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24

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283

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2008-04-11T00:00:00Z

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18408007

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2423239

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