Pathophysiology of Migraine: A Disorder of Sensory Processing. - Wikidata - awgldk - TriplyDB

Pathophysiology of Migraine: A Disorder of Sensory Processing.

Pathophysiology of Migraine: A Disorder of Sensory Processing.

http://www.wikidata.org/entity/Q39127073

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Differential inhibitory response to telcagepant on αCGRP induced vasorelaxation and intracellular Ca2+ levels in the perfused and non-perfused isolated rat middle cerebral artery.Comparative effects of traditional Chinese and Western migraine medicines in an animal model of nociceptive trigeminovascular activation.Initiation of spreading depression by synaptic and network hyperactivity: Insights into trigger mechanisms of migraine aura.Premonitory Symptoms of Migraine in Childhood and Adolescence.Hopes for the Future of Pain Control.An update on migraine: current understanding and future directions.The Trigeminovascular Pathway: Role of CGRP and CGRP Receptors in Migraine.Anti-CGRP Monoclonal Antibodies: the Next Era of Migraine Prevention?Assessment of gray and white matter structural alterations in migraineurs without aura.Interactions between the Kynurenine and the Endocannabinoid System with Special Emphasis on Migraine The Molecular Fingerprint of Dorsal Root and Trigeminal Ganglion Neurons.Triptans and CGRP blockade - impact on the cranial vasculature.Topiramate modulates trigeminal pain processing in thalamo-cortical networks in humans after single dose administration Estrogen-Dependent Changes in Dura Mater Microvasculature Add New Insights to the Pathogenesis of Headache.Targeting the central projection of the dural trigeminovascular system for migraine prophylaxis.Daith Piercing in a Case of Chronic Migraine: A Possible Vagal Modulation.The Posterior Insula Shows Disrupted Brain Functional Connectivity in Female Migraineurs Without Aura Based on Brainnetome Atlas.Side effects associated with current and prospective antimigraine pharmacotherapies.Treatment of disabling headache with greater occipital nerve injections in a large population of childhood and adolescent patients: a service evaluation.Anti-migraine effect of ∆9-tetrahydrocannabinol in the female rat.Glutamate receptor antagonists with the potential for migraine treatment.Dysautonomia in the pathogenesis of migraine.Botulinum Toxin A for the Treatment of Photophobia and Dry Eye.Intranasally administered IGF-1 inhibits spreading depression in vivo.Neurovascular mechanisms of migraine and cluster headache.Inhibition of toll-like receptor 4 alleviates hyperalgesia induced by acute dural inflammation in experimental migraine.Distribution of CGRP and CGRP receptor components in the rat brain.Soluble guanylyl cyclase is a critical regulator of migraine-associated pain.Migraine Treatment: Current Acute Medications and Their Potential Mechanisms of Action.Targeted Orexin and Hypothalamic Neuropeptides for Migraine.Calcitonin gene-related peptide (receptor) antibodies: an exciting avenue for migraine treatment.Alterations in post-movement beta event related synchronization throughout the migraine cycle: A controlled, longitudinal study.Migraine Therapy: Current Approaches and New Horizons.The Biology of Monoclonal Antibodies: Focus on Calcitonin Gene-Related Peptide for Prophylactic Migraine Therapy.Inhibitory Gi/O-coupled receptors in somatosensory neurons: Potential therapeutic targets for novel analgesics.PACAP and migraine headache: immunomodulation of neural circuits in autonomic ganglia and brain parenchyma.Glutamate and Its Receptors as Therapeutic Targets for Migraine.Targeted 5-HT1F Therapies for Migraine.Current Prophylactic Medications for Migraine and Their Potential Mechanisms of Action.Targeted CGRP Small Molecule Antagonists for Acute Migraine Therapy.

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P2860

Pathophysiology of Migraine: A Disorder of Sensory Processing.

http://www.wikidata.org/entity/Q39127073

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Pathophysiology of Migraine: A Disorder of Sensory Processing.

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Christoph Schankin

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Jan Hoffmann

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Margarida Martins-Oliveira

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P2860

Short sleep duration is associated with reduced leptin, elevated ghrelin, and increased body mass index

A role of melanin-concentrating hormone producing neurons in the central regulation of paradoxical sleep

Impact of sleep debt on metabolic and endocrine function

Modeling of a human circadian mutation yields insights into clock regulation by PER2

Haploinsufficiency of ATP1A2 encoding the Na+/K+ pump alpha2 subunit associated with familial hemiplegic migraine type 2

Orexins and orexin receptors: a family of hypothalamic neuropeptides and G protein-coupled receptors that regulate feeding behavior

RAMPs regulate the transport and ligand specificity of the calcitonin-receptor-like receptor

Familial hemiplegic migraine mutations increase Ca(2+) influx through single human CaV2.1 channels and decrease maximal CaV2.1 current density in neurons.

Nocturnal rise of leptin in lean, obese, and non-insulin-dependent diabetes mellitus subjects

Occipital nerve stimulation for the treatment of intractable chronic migraine headache: ONSTIM feasibility study

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