about
Restoration of oligodendrocyte pools in a mouse model of chronic cerebral hypoperfusionNeuroendocrine signaling modulates specific neural networks relevant to migraineGliovascular disruption and cognitive deficits in a mouse model with features of small vessel disease.Mechanically-induced cortical spreading depression associated regional cerebral blood flow changes are blocked by Na+ ion channel blockade.Diencephalic and brainstem mechanisms in migraine.A translational in vivo model of trigeminal autonomic cephalalgias: therapeutic characterization.Transcranial magnetic stimulation and potential cortical and trigeminothalamic mechanisms in migraine.Animal models of migraine: looking at the component parts of a complex disorder.The hypothalamic orexinergic system: pain and primary headaches.Cluster headache, hypothalamus, and orexin.Modulation of trigeminovascular processing: novel insights into primary headache disorders.Headache and sleep: shared pathophysiological mechanisms.Current and novel insights into the neurophysiology of migraine and its implications for therapeutics.Pathophysiology of Migraine: A Disorder of Sensory Processing.Chronic cerebral hypoperfusion alters amyloid-β peptide pools leading to cerebral amyloid angiopathy, microinfarcts and haemorrhages in Tg-SwDI mice.Dimethyl fumarate improves white matter function following severe hypoperfusion: Involvement of microglia/macrophages and inflammatory mediators.Hypertension fails to disrupt white matter integrity in young or aged Fisher (F44) Cyp1a1Ren2 transgenic ratsModulation of nociceptive dural input to the trigeminocervical complex through GluK1 kainate receptors.Cortical spreading depression-associated cerebral blood flow changes induced by mechanical stimulation are modulated by AMPA and GABA receptors.Neurons of the dopaminergic/calcitonin gene-related peptide A11 cell group modulate neuronal firing in the trigeminocervical complex: an electrophysiological and immunohistochemical study.Orexin 1 receptor activation attenuates neurogenic dural vasodilation in an animal model of trigeminovascular nociception.The role of the brainstem in migraine: Potential brainstem effects of CGRP and CGRP receptor activation in animal models.Minocycline reduces microgliosis and improves subcortical white matter function in a model of cerebral vascular disease.Acid-sensing ion channel 1: a novel therapeutic target for migraine with aura.Involvement of corticotrophin-releasing factor and orexin-A in chronic migraine and medication overuse headache: findings from cerebrospinal fluid.Modulation of nociceptive dural input to the trigeminal nucleus caudalis via activation of the orexin 1 receptor in the rat.Occipital afferent activation of second order neurons in the trigeminocervical complex in rat.Controlled hypertension induces cerebrovascular and gene alterations in Cyp1a1-Ren2 transgenic rats.Migraine Therapy: Current Approaches and New Horizons.PACAP in hypothalamic regulation of sleep and circadian rhythm: importance for headache.MRI is a sensitive marker of subtle white matter pathology in hypoperfused miceDivergent influences of the locus coeruleus on migraine pathophysiologyNitroglycerine triggers triptan-responsive cranial allodynia and trigeminal neuronal hypersensitivityNeurobiology of migraineForeword: The CGRP Pathway and Migraine Prevention: Reducing the Burden of DiseaseRapid uptake of sumatriptan into the brain: An ongoing question of blood-brain barrier permeabilityAn Update: Pathophysiology of MigraineAcid-sensing ion channel 3 blockade inhibits durovascular and nitric oxide-mediated trigeminal painAnimal models of migraine and experimental techniques used to examine trigeminal sensory processing
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Philip R Holland
@nl
Philip R Holland
@sl
Philip R. Holland
@en
Philip R. Holland
@es
type
label
Philip R Holland
@nl
Philip R Holland
@sl
Philip R. Holland
@en
Philip R. Holland
@es
prefLabel
Philip R Holland
@nl
Philip R Holland
@sl
Philip R. Holland
@en
Philip R. Holland
@es
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