Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation. - Wikidata - awgldk - TriplyDB

Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.

Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.

http://www.wikidata.org/entity/Q39149749

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Mechanisms of nerve growth factor signaling in bone nociceptors and in an animal model of inflammatory bone pain.SDF1-CXCR4 signaling contributes to persistent pain and hypersensitivity via regulating excitability of primary nociceptive neurons: involvement of ERK-dependent Nav1.8 up-regulation.Topical ambroxol for the treatment of neuropathic pain. An initial clinical observation.TLR signaling adaptor protein MyD88 in primary sensory neurons contributes to persistent inflammatory and neuropathic pain and neuroinflammation.Effect of TRPV4-p38 MAPK Pathway on Neuropathic Pain in Rats with Chronic Compression of the Dorsal Root Ganglion Upregulation of the sodium channel NaVβ4 subunit and its contributions to mechanical hypersensitivity and neuronal hyperexcitability in a rat model of radicular pain induced by local dorsal root ganglion inflammation.Functional inhibition of chemokine receptor CCR2 by dicer-substrate-siRNA prevents pain development Osteoarthritis-dependent changes in antinociceptive action of Nav1.7 and Nav1.8 sodium channel blockers: An in vivo electrophysiological study in the rat.α-lipoic acid suppresses neuronal excitability and attenuates colonic hypersensitivity to colorectal distention in diabetic rats.Ambroxol for the treatment of fibromyalgia: science or fiction?[Topical ambroxol for the treatment of neuropathic pain: A first clinical observation. German version].Functional up-regulation of Nav1.8 sodium channel on dorsal root ganglia neurons contributes to the induction of scorpion sting pain.The Na1.7 Channel Subtype as an Antinociceptive Target for Spider Toxins in Adult Dorsal Root Ganglia Neurons

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P2860

Functional up-regulation of Nav1.8 sodium channel in Aβ afferent fibers subjected to chronic peripheral inflammation.

http://www.wikidata.org/entity/Q39149749

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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name

Functional up-regulation of Na ...... ronic peripheral inflammation.

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Functional up-regulation of Na ...... ronic peripheral inflammation.

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type

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Functional up-regulation of Na ...... ronic peripheral inflammation.

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Functional up-regulation of Na ...... ronic peripheral inflammation.

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Functional up-regulation of Na ...... ronic peripheral inflammation.

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1742-2094-11-45

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Journal of Neuroinflammation

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Functional up-regulation of Na ...... hronic peripheral inflammation

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Ahmed Chraibi

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Marc-André Dansereau

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Michael Biet

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Mounir Belkouch

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Philippe Sarret

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Robert Dumaine

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P2860

Synaptic reorganization in the substantia gelatinosa after peripheral nerve neuroma formation: aberrant innervation of lamina II neurons by Abeta afferents

Neuronal circuitry for pain processing in the dorsal horn

Potent spinal analgesia elicited through stimulation of NTS2 neurotensin receptors

Sodium currents of large (Abeta-type) adult cutaneous afferent dorsal root ganglion neurons display rapid recovery from inactivation before and after axotomy

Relationship of axonal voltage-gated sodium channel 1.8 (NaV1.8) mRNA accumulation to sciatic nerve injury-induced painful neuropathy in rats

Contactin regulates the current density and axonal expression of tetrodotoxin-resistant but not tetrodotoxin-sensitive sodium channels in DRG neurons

Distribution of the tetrodotoxin-resistant sodium channel PN3 in rat sensory neurons in normal and neuropathic conditions.

Bilateral downregulation of Nav1.8 in dorsal root ganglia of rats with bone cancer pain induced by inoculation with Walker 256 breast tumor cells

A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain.

Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia

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1742-2094-11-45

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