SDF1-CXCR4 signaling contributes to persistent pain and hypersensitivity via regulating excitability of primary nociceptive neurons: involvement of ERK-dependent Nav1.8 up-regulation.
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Down-Regulation of CXCL12/CXCR4 Expression Alleviates Ischemia-Reperfusion-Induced Inflammatory Pain via Inhibiting Glial TLR4 Activation in the Spinal Cord.Up-regulation of CXCR4 expression contributes to persistent abdominal pain in rats with chronic pancreatitis.Chemokine receptor CXCR4 regulates CaMKII/CREB pathway in spinal neurons that underlies cancer-induced bone pain.[EXPRESS] CXCL12/CXCR4 signaling mediated ERK1/2 activation in spinal cord contributes to the pathogenesis of postsurgical pain in rats.Effect of CXCL12/CXCR4 signaling on neuropathic pain after chronic compression of dorsal root ganglionOverexpression of GRK6 attenuates neuropathic pain via suppression of CXCR2 in rat dorsal root ganglion.Cytokine cascades induced by mechanical trauma injury alter voltage-gated sodium channel activity in intact cortical neurons.Macrophage-derived HMGB1 as a Pain Mediator in the Early Stage of Acute Pancreatitis in Mice: Targeting RAGE and CXCL12/CXCR4 Axis.SDF1-CXCR4 Signaling Contributes to the Transition from Acute to Chronic Pain State.SDF1-CXCR4 Signaling Maintains Central Post-Stroke Pain through Mediation of Glial-Neuronal InteractionsCXCR4 antagonist AMD3100 elicits analgesic effect and restores the GlyRα3 expression against neuropathic pain.Ambroxol for the treatment of fibromyalgia: science or fiction?Upregulation of CX3CL1 mediated by NF-κB activation in dorsal root ganglion contributes to peripheral sensitization and chronic pain induced by oxaliplatin administration.Moxibustion eases chronic inflammatory visceral pain through regulating MEK, ERK and CREB in rats.Activation of STAT3-mediated CXCL12 up-regulation in the dorsal root ganglion contributes to oxaliplatin-induced chronic pain.A Novel Role for Lymphotactin (XCL1) Signaling in the Nervous System: XCL1 Acts via its Receptor XCR1 to Increase Trigeminal Neuronal Excitability.Modulation of Gq-Rho Signaling by the ERK MAPK Pathway Controls Locomotion in Caenorhabditis elegans.Reducing CXCR4-mediated nociceptor hyperexcitability reverses painful diabetic neuropathy.
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P2860
SDF1-CXCR4 signaling contributes to persistent pain and hypersensitivity via regulating excitability of primary nociceptive neurons: involvement of ERK-dependent Nav1.8 up-regulation.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
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2015年學術文章
@zh-hant
name
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@ast
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@en
type
label
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@ast
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@en
prefLabel
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@ast
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@en
P2093
P2860
P1476
SDF1-CXCR4 signaling contribut ...... ependent Nav1.8 up-regulation.
@en
P2093
Chun-Li Li
Xiao-Liang Wang
P2860
P2888
P356
10.1186/S12974-015-0441-2
P577
2015-11-24T00:00:00Z
P5875
P6179
1048856628