Malignant melanoma cells acquire resistance to DNA interstrand cross-linking chemotherapeutics by p53-triggered upregulation of DDB2/XPC-mediated DNA repair.
about
Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptationMDM2 is an important prognostic and predictive factor for platin-pemetrexed therapy in malignant pleural mesotheliomas and deregulation of P14/ARF (encoded by CDKN2A) seems to contribute to an MDM2-driven inactivation of P53.Transcriptional and Posttranslational Regulation of Nucleotide Excision Repair: The Guardian of the Genome against Ultraviolet RadiationExposure of Human Lung Cells to Tobacco Smoke Condensate Inhibits the Nucleotide Excision Repair PathwayMicroRNAs Used in Combination with Anti-Cancer Treatments Can Enhance Therapy EfficacyThe role of the cancer stem cell marker CD271 in DNA damage response and drug resistance of melanoma cells.Mechanistic insights into the impact of Cold Atmospheric Pressure Plasma on human epithelial cell lines.Chromatin retention of DNA damage sensors DDB2 and XPC through loss of p97 segregase causes genotoxicity.NRIP/DCAF6 stabilizes the androgen receptor protein by displacing DDB2 from the CUL4A-DDB1 E3 ligase complex in prostate cancer.Attenuated NER expressions of XPF and XPC associated with smoking are involved in the recurrence of bladder cancer.Targeting MPS1 Enhances Radiosensitization of Human Glioblastoma by Modulating DNA Repair ProteinsFaster DNA Repair of Ultraviolet-Induced Cyclobutane Pyrimidine Dimers and Lower Sensitivity to Apoptosis in Human Corneal Epithelial Cells than in Epidermal Keratinocytes.JWA reverses cisplatin resistance via the CK2-XRCC1 pathway in human gastric cancer cells.SIRT1 activation mediates heat-induced survival of UVB damaged KeratinocytesInvestigating the different mechanisms of genotoxic and non-genotoxic carcinogens by a gene set analysis.Chemotherapy Resistance Mechanisms in Advanced Skin Cancer.Apoptosis induced by temozolomide and nimustine in glioblastoma cells is supported by JNK/c-Jun-mediated induction of the BH3-only protein BIM.Inorganic arsenic inhibits the nucleotide excision repair pathway and reduces the expression of XPCAccumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death.Heme oxygenase-1 plays a crucial role in chemoresistance in acute myeloid leukemia.B-Raf inhibitor vemurafenib in combination with temozolomide and fotemustine in the killing response of malignant melanoma cells.The multifaceted influence of histone deacetylases on DNA damage signalling and DNA repair.Veliparib in combination with whole brain radiation therapy in patients with brain metastases: results of a phase 1 study.A DDB2 mutant protein unable to interact with PCNA promotes cell cycle progression of human transformed embryonic kidney cells.Melanoma cells replicate through chemotherapy by reducing levels of key homologous recombination protein RAD51 and increasing expression of translesion synthesis DNA polymerase ζ.Tyrosine Kinase Inhibitors Protect the Salivary Gland from Radiation Damage by Inhibiting Activation of Protein Kinase C-δ.Strong antitumor synergy between DNA crosslinking and HSP90 inhibition causes massive premitotic DNA fragmentation in ovarian cancer cells.
P2860
Q27026117-57ADC2A1-C62D-433C-932D-0DC2DE462228Q27853125-D6EA502D-16F3-45E6-B79B-6BFBF7B6F046Q28070128-33EE6771-8C15-4DEF-B381-77BB284B8828Q28552525-3719B610-D3C2-4071-B0EE-BF212058EF05Q28829013-713EB603-EBFD-45A9-B65C-1D6E68C2D9F5Q30837718-4D8EF130-A837-413F-AF74-5D24185696B4Q33469694-FF4CF943-17C2-41CE-95DA-D44121AD92F8Q33557027-8A3CB85F-E6F1-43CE-ABC3-5527B9288085Q33591581-741E3E6F-9E1C-4C82-83A5-B178CF300842Q34764712-C5D4B106-A598-4D42-AD42-DF93A091AEF1Q35611529-269B4839-05AE-4BE0-8103-73B831A64B5FQ36128004-FA159227-9B26-4D24-99F7-9C33F658E871Q36291991-9B133B92-2ECB-4A0E-BE01-31381B4457CCQ36397437-992D8DF5-AC84-4572-AB5F-693215EDC9FAQ37538175-C7039CE5-D5B5-4AD0-B652-C9C3D958DE12Q37736532-DF270D0F-B5E1-4F11-BB49-AEB28B5F8173Q38261426-C41A05AA-856E-4AC8-8D4B-348B0F70E2DAQ38288940-64E711E0-22FA-40BF-A9AB-D0663CC8C7FBQ38754109-8C63DE86-0C3E-4015-83DE-842F6B190181Q38848661-88E9D663-0484-48FB-B68A-427068FA840CQ38923467-38BC26F8-072F-4B8F-AF76-FFA958415FC2Q38979892-6DCCA258-065A-41C0-9BD9-AEE0C08FCAE4Q39038900-ED0B9C8B-B62D-4038-82BC-A0C71A998D03Q39987578-35617FF5-2DAA-4D63-BF5A-EC35C8092E84Q47139347-ED356D32-46B4-44F9-9303-F95E752704FEQ50931131-8D032C5E-6AA8-489C-B7C9-DE1D1534DCDFQ52676453-C1201594-E23C-4DB3-9838-902DE50CFC85
P2860
Malignant melanoma cells acquire resistance to DNA interstrand cross-linking chemotherapeutics by p53-triggered upregulation of DDB2/XPC-mediated DNA repair.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@en
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@nl
type
label
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@en
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@nl
prefLabel
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@en
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@nl
P2093
P2860
P356
P1433
P1476
Malignant melanoma cells acqui ...... DDB2/XPC-mediated DNA repair.
@en
P2093
C Barckhausen
S C Naumann
P2860
P2888
P304
P356
10.1038/ONC.2013.141
P407
P577
2013-04-22T00:00:00Z
P6179
1037053450