NR5A2 discovering compounds that block tumor growth in PDAC.

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Accelerating progress in the fight against pancreatic cancer Proceedings of the 2017 Leo and Anne Albert Symposium for Pancreatic Cancer Research.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC.

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NR5A2 discovering compounds that block tumor growth in PDAC

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P2093

Robert Fletterick

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A genome-wide association study identifies pancreatic cancer susceptibility loci on chromosomes 13q22.1, 1q32.1 and 5p15.33

Structural basis for ligand-independent activation of the orphan nuclear receptor LRH-1

Structural basis of coactivation of liver receptor homolog-1 by -catenin

Disulfide-Trapping Identifies a New, Effective Chemical Probe for Activating the Nuclear Receptor Human LRH-1 (NR5A2)

Canonical Wnt/β-catenin regulation of liver receptor homolog-1 mediates pluripotency gene expression

Identification of small molecule agonists of the orphan nuclear receptors liver receptor homolog-1 and steroidogenic factor-1.

Nuclear receptor liver receptor homologue 1 (LRH-1) regulates pancreatic cancer cell growth and proliferation.

Structure of Liver Receptor Homolog-1 (NR5A2) with PIP3 hormone bound in the ligand binding pocket.

Structure-based discovery of antagonists of nuclear receptor LRH-1.

Molecular basis for the regulation of the nuclear receptor LRH-1.

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89-93

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scholarly article

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10.1002/JSO.24639

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116

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Robert Fletterick

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2017-04-26T00:00:00Z

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28445593

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5519325

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NR5A2 discovering compounds that block tumor growth in PDAC.

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P2860

P2860

NR5A2 discovering compounds that block tumor growth in PDAC.

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