The genesis and unique properties of the lymphovascular tumor embolus are because of calpain-regulated proteolysis of E-cadherin.
about
Cleavage of E-cadherin and β-catenin by calpain affects Wnt signaling and spheroid formation in suspension cultures of human pluripotent stem cellsMinus end-directed motor KIFC3 suppresses E-cadherin degradation by recruiting USP47 to adherens junctions.Spherical cancer models in tumor biology.Capn4 is a marker of poor clinical outcomes and promotes nasopharyngeal carcinoma metastasis via nuclear factor-κB-induced matrix metalloproteinase 2 expressionThe calpain system is associated with survival of breast cancer patients with large but operable inflammatory and non-inflammatory tumours treated with neoadjuvant chemotherapy.The VE-cadherin cytoplasmic domain undergoes proteolytic processing during endocytosis.Meta-analysis of downregulated E-cadherin as a poor prognostic biomarker for cervical cancer.E-Cadherin fragments as potential mediators for peritoneal metastasis in advanced epithelial ovarian cancerSingular v dual inhibition of SNF2L and its isoform, SNF2LT, have similar effects on DNA damage but opposite effects on the DNA damage response, cancer cell growth arrest and apoptosis.Comparison of the protein expression of calpain-1, calpain-2, calpastatin and calmodulin between gastric cancer and normal gastric mucosa.Defective Protein Catabolism in Atherosclerotic Vascular Inflammation.CRISPR-mediated deletion of the PECAM-1 cytoplasmic domain increases receptor lateral mobility and strengthens endothelial cell junctional integrity.Use of αv Integrin Linked to GFP to Image Molecular Dynamics in Trafficking Cancer-Cell Emboli.A Model for Link Pruning to Establish Correctly Polarized and Oriented Tip Links in Hair Bundles.Gene/protein expression of CAPN1/2-CAST system members is associated with ERK1/2 kinases activity as well as progression and clinical outcome in human laryngeal cancer.Significance and prognosis of epithelial-cadherin expression in invasive breast carcinoma.
P2860
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P2860
The genesis and unique properties of the lymphovascular tumor embolus are because of calpain-regulated proteolysis of E-cadherin.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@en
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@nl
type
label
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@en
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@nl
prefLabel
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@en
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@nl
P2093
P2860
P356
P1433
P1476
The genesis and unique propert ...... ted proteolysis of E-cadherin.
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2012.180
P407
P50
P577
2012-05-14T00:00:00Z