about
Cardiac-specific, inducible ClC-3 gene deletion eliminates native volume-sensitive chloride channels and produces myocardial hypertrophy in adult miceOrai1 interacts with STIM1 and mediates capacitative Ca2+ entry in mouse pulmonary arterial smooth muscle cells.Regulation of nuclear TDP-43 by NR2A-containing NMDA receptors and PTEN.Early to intermediate steps of tumor embolic formation involve specific proteolytic processing of E-cadherin regulated by Rab7.The genesis and unique properties of the lymphovascular tumor embolus are because of calpain-regulated proteolysis of E-cadherin.TRPC1 and Orai1 interact with STIM1 and mediate capacitative Ca(2+) entry caused by acute hypoxia in mouse pulmonary arterial smooth muscle cells.Targeted inactivation of cystic fibrosis transmembrane conductance regulator chloride channel gene prevents ischemic preconditioning in isolated mouse heart.Cardiac-specific overexpression of the human short CLC-3 chloride channel isoform in mice.Upregulation of profilin, cofilin-2 and LIMK2 in cultured pulmonary artery smooth muscle cells and in pulmonary arteries of monocrotaline-treated rats.
P50
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P50
description
researcher
@en
հետազոտող
@hy
name
Honglin Tian
@ast
Honglin Tian
@en
Honglin Tian
@es
Honglin Tian
@nl
type
label
Honglin Tian
@ast
Honglin Tian
@en
Honglin Tian
@es
Honglin Tian
@nl
prefLabel
Honglin Tian
@ast
Honglin Tian
@en
Honglin Tian
@es
Honglin Tian
@nl
P69
P1153
7202161993
P2038
Honglin_Tian