NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

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The role of ubiquitin ligases in cardiac disease.

P2860

Q38164901-ECA17384-FF62-4821-869E-737ECEDF469C

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NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

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http://www.wikidata.org/entity/Q39388938

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2013 nî lūn-bûn

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2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh

2013年學術文章

@zh-hant

name

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@en

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@nl

type

Item

label

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@en

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@nl

prefLabel

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@en

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

@nl

P1476

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy

@en

P2093

Diana Shy

http://www.w3.org/2001/XMLSchema#string

P2860

SAP97 and dystrophin macromolecular complexes determine two pools of cardiac sodium channels Nav1.5 in cardiomyocytes

Interactions between ankyrin-G, Plakophilin-2, and Connexin43 at the cardiac intercalated disc

Loss of plakophilin-2 expression leads to decreased sodium current and slower conduction velocity in cultured cardiac myocytes

Diagnosis of arrhythmogenic right ventricular cardiomyopathy/dysplasia: proposed modification of the Task Force Criteria

A red-shifted Renilla luciferase for transient reporter-gene expression.

Zonula occludens-1 alters connexin43 gap junction size and organization by influencing channel accretion

Preliminary report: effect of encainide and flecainide on mortality in a randomized trial of arrhythmia suppression after myocardial infarction.

The perinexus: a new feature of Cx43 gap junction organization.

Reduced heterogeneous expression of Cx43 results in decreased Nav1.5 expression and reduced sodium current that accounts for arrhythmia vulnerability in conditional Cx43 knockout mice.

Dynamic reciprocity of sodium and potassium channel expression in a macromolecular complex controls cardiac excitability and arrhythmia

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467-470

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scholarly article

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10.2217/FCA.13.38

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P478

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Hugues Abriel

Ludovic Gillet

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2013-07-01T00:00:00Z

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247770312

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23834686

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NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

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P2860

P2860

NaV1.5 and interacting proteins in human arrhythmogenic cardiomyopathy.

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type

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698