Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes.
about
Reduced up-regulation of the nitric oxide pathway and impaired endothelial and smooth muscle functions in the female type 2 diabetic goto-kakizaki rat heartMacrophage plasticity and polarization: in vivo veritasFinding type 2 diabetes causal single nucleotide polymorphism combinations and functional modules from genome-wide association data.Topical Naltrexone as Treatment for Type 2 Diabetic Cutaneous WoundsEffect of plant extracts on H2O2-induced inflammatory gene expression in macrophages.FOXO1, TGF-β regulation and wound healingUpregulation of CREM/ICER suppresses wound endothelial CRE-HIF-1α-VEGF-dependent signaling and impairs angiogenesis in type 2 diabetes.Dehydroabietic acid reverses TNF-α-induced the activation of FOXO1 and suppression of TGF-β1/Smad signaling in human adult dermal fibroblasts.Full-Thickness Thermal Injury Delays Wound Closure in a Murine ModelFOXO1 differentially regulates both normal and diabetic wound healing.LXA4 actions direct fibroblast function and wound closureThe GSK-3β/Fyn/Nrf2 pathway in fibroblasts and wounds of type 2 diabetes: On the road to an evidence-based therapy of non-healing wounds.NLRP3 Inflammasome Expression and Signaling in Human Diabetic Wounds and in High Glucose Induced Macrophages.Myeloid cell expressed proprotein convertase FURIN attenuates inflammation.Redox Signaling in Diabetic Wound Healing Regulates Extracellular Matrix Deposition.Diabetic Foot Ulcers and Epidermal Growth Factor: Revisiting the Local Delivery Route for a Successful Outcome.Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.Topical naltrexone accelerates full-thickness wound closure in type 1 diabetic rats by stimulating angiogenesis.Selective blockade of estrogen receptor beta improves wound healing in diabetes.Bee Venom Accelerates Wound Healing in Diabetic Mice by Suppressing Activating Transcription Factor-3 (ATF-3) and Inducible Nitric Oxide Synthase (iNOS)-Mediated Oxidative Stress and Recruiting Bone Marrow-Derived Endothelial Progenitor Cells.Transforming growth factor-β genetic polymorphisms on development of liver cirrhosis in a meta-analysis.20(S)-Protopanaxadiol enhances angiogenesis via HIF-1α-mediated VEGF secretion by activating p70S6 kinase and benefits wound healing in genetically diabetic mice.Chronic treatment with the glucocorticoid receptor antagonist RU486 inhibits diabetes-induced enhancement of experimental periodontitis.Notoginsenoside Ft1 Promotes Fibroblast Proliferation via PI3K/Akt/mTOR Signaling Pathway and Benefits Wound Healing in Genetically Diabetic Mice.
P2860
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P2860
Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
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2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
@zh-hant
name
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@en
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@nl
type
label
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@en
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@nl
prefLabel
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@en
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@nl
P2860
P356
P1433
P1476
Impaired TGF-β signaling and a ...... rat model of type 2 diabetes.
@en
P2093
Issam M Francis
Milad S Bitar
P2860
P304
P356
10.1039/C0MB00317D
P577
2011-08-18T00:00:00Z