Delayed increase of tyrosine hydroxylase expression in rat nigrostriatal system after traumatic brain injury. - Wikidata - awgldk - TriplyDB

Delayed increase of tyrosine hydroxylase expression in rat nigrostriatal system after traumatic brain injury.

Delayed increase of tyrosine hydroxylase expression in rat nigrostriatal system after traumatic brain injury.

http://www.wikidata.org/entity/Q39494122

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Catecholamines and cognition after traumatic brain injury Sex differences in mitochondrial (dys)function: Implications for neuroprotection Imaging brain plasticity after trauma.Persistent cognitive dysfunction after traumatic brain injury: A dopamine hypothesis The effects of piroxicam in the attenuation of MPP+/MPTP toxicity in vitro and in vivo.Experimental traumatic brain injury alters ethanol consumption and sensitivity.Does traumatic brain injury increase risk for substance abuse?Methamphetamine potentiates behavioral and electrochemical responses after mild traumatic brain injury in mice Biologic and plastic effects of experimental traumatic brain injury treatment paradigms and their relevance to clinical rehabilitation.Targeting Dopamine in Acute Traumatic Brain Injury Substantia nigra vulnerability after a single moderate diffuse brain injury in the rat.Effects of nicotine administration on striatal dopamine signaling after traumatic brain injury in rats.The effect of environmental enrichment on substantia nigra gene expression after traumatic brain injury in rats.Associations of proteins relevant to MAPK signaling pathway (p38MAPK-1,HIF-1 and HO-1) with coronary lesion characteristics and prognosis of peri-menopausal women.Consequences of developmental exposure to concentrated ambient ultrafine particle air pollution combined with the adult paraquat and maneb model of the Parkinson's disease phenotype in male mice.Traumatic brain injuries during development disrupt dopaminergic signaling.Ventricular fibrillation cardiac arrest produces a chronic striatal hyperdopaminergic state that is worsened by methylphenidate treatment.Factors affecting increased risk for substance use disorders following traumatic brain injury: What we can learn from animal models.A study on the mechanism by which MDMA protects against dopaminergic dysfunction after minimal traumatic brain injury (mTBI) in mice.Differential effect of traumatic brain injury on the nuclear factor of activated T Cells C3 and C4 isoforms in the rat hippocampus Adolescent Traumatic Brain Injury Induces Chronic Mesolimbic Neuroinflammation with Concurrent Enhancement in the Rewarding Effects of Cocaine in Mice during Adulthood.Traumatic brain injury reduces striatal tyrosine hydroxylase activity and potassium-evoked dopamine release in rats Normabaric Hyperoxia Treatment Improved Locomotor Activity of C57BL/6J Mice through Enhancing Dopamine Genes Following Fluid-Percussion Injury in Striatum.Controlled cortical impact injury influences methylphenidate-induced changes in striatal dopamine neurotransmission.Chronic methylphenidate treatment enhances striatal dopamine neurotransmission after experimental traumatic brain injury.Lateral Fluid Percussion Injury Impairs Hippocampal Synaptic Soluble N-Ethylmaleimide Sensitive Factor Attachment Protein Receptor Complex Formation.The dopamine and cAMP regulated phosphoprotein, 32 kDa (DARPP-32) signaling pathway: a novel therapeutic target in traumatic brain injury.Impact of Traumatic Brain Injury on Dopaminergic Transmission.Proteomic analysis and brain-specific systems biology in a rodent model of penetrating ballistic-like brain injury.Normobaric hyperoxia treatment prevents early alteration in dopamine level in mice striatum after fluid percussion injury: a biochemical approach.Executive (dys)function after traumatic brain injury: special considerations for behavioral pharmacology

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P2860

Delayed increase of tyrosine hydroxylase expression in rat nigrostriatal system after traumatic brain injury.

http://www.wikidata.org/entity/Q39494122

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

@zh-my

2006年学术文章

@zh-sg

2006年學術文章

@yue

2006年學術文章

@zh

2006年學術文章

@zh-hant

name

Delayed increase of tyrosine h ...... after traumatic brain injury.

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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type

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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J.BRAINRES.2006.11.087

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Delayed increase of tyrosine h ...... after traumatic brain injury.

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C Edward Dixon

http://www.w3.org/2001/XMLSchema#string

Hong Qu Yan

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Lifang Shao

http://www.w3.org/2001/XMLSchema#string

Xiangbai Chen

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Xiecheng Ma

http://www.w3.org/2001/XMLSchema#string

Youming Li

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P2860

Antiproliferative proteins of the BTG/Tob family are degraded by the ubiquitin-proteasome system

Protection and repair of the nigrostriatal dopaminergic system by GDNF in vivo

Results of the NIH consensus conference on "rehabilitation of persons with traumatic brain injury".

Progressive parkinsonism in boxers.

Multiple parallel memory systems in the brain of the rat.

Neuropharmacology of TBI-induced plasticity.

Parkinson's syndrome after closed head injury: a single case report

Working memory deficits after traumatic brain injury: catecholaminergic mechanisms and prospects for treatment -- a review.

Neuroprotection with the proteasome inhibitor MLN519 in focal ischemic brain injury: relation to nuclear factor kappaB (NF-kappaB), inflammatory gene expression, and leukocyte infiltration.

Immunohistochemical studies on the localization and distribution of monoamine neuron systems in the rat brain. I. Tyrosine hydroxylase in the mes- and diencephalon.

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traumatic brain injury

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