OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
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Crossing the line: selection and evolution of virulence traitsQuantitative mass spectrometry catalogues Salmonella pathogenicity island-2 effectors and identifies their cognate host binding partnersSalmonella pathogenicity island 2 is expressed prior to penetrating the intestine.Regulatory principles governing Salmonella and Yersinia virulenceBacterial regulon evolution: distinct responses and roles for the identical OmpR proteins of Salmonella Typhimurium and Escherichia coli in the acid stress responseTransient requirement of the PrrA-PrrB two-component system for early intracellular multiplication of Mycobacterium tuberculosisExpression of the AcrAB Components of the AcrAB-TolC Multidrug Efflux Pump of Yersinia enterocolitica Is Subject to Dual Regulation by OmpROmpR controls Yersinia enterocolitica motility by positive regulation of flhDC expressionMolecular basis of bacterial outer membrane permeability revisitedSalmonella trafficking is defined by continuous dynamic interactions with the endolysosomal system.Flagella facilitate escape of Salmonella from oncotic macrophages.Examination of Salmonella gene expression in an infected mammalian host using the green fluorescent protein and two-colour flow cytometry.Systematic analysis of the regulation of type three secreted effectors in Salmonella enterica serovar Typhimurium.Coordinated regulation of virulence during systemic infection of Salmonella enterica serovar Typhimurium.RNA arbitrarily primed PCR survey of genes regulated by ToxR in the deep-sea bacterium Photobacterium profundum strain SS9.Systems level analysis of two-component signal transduction systems in Erwinia amylovora: role in virulence, regulation of amylovoran biosynthesis and swarming motility.Control of Salmonella pathogenicity island-2 gene expression.Salmonella pathogenicity island 1-independent induction of apoptosis in infected macrophages by Salmonella enterica serotype typhimurium.The SdiA-regulated gene srgE encodes a type III secreted effector.The acetate switch.S. Typhimurium sseJ gene decreases the S. Typhi cytotoxicity toward cultured epithelial cells.Comparative proteomic analysis of Salmonella enterica serovar Typhimurium ppGpp-deficient mutant to identify a novel virulence protein required for intracellular survival in macrophagesSignal transduction pathway mediated by the novel regulator LoiA for low oxygen tension induced Salmonella Typhimurium invasion.SlyA regulates function of Salmonella pathogenicity island 2 (SPI-2) and expression of SPI-2-associated genes.Regulation of a type III and a putative secretion system in Edwardsiella tarda by EsrC is under the control of a two-component system, EsrA-EsrBHierarchical control of anaerobic gene expression in Escherichia coli K-12: the nitrate-responsive NarX-NarL regulatory system represses synthesis of the fumarate-responsive DcuS-DcuR regulatory system.Evolution of two-component signal transduction systemsThe Salmonella enterica PhoP directly activates the horizontally acquired SPI-2 gene sseL and is functionally different from a S. bongori ortholog.Salmonella pathogenicity island 2.Unravelling the mysteries of virulence gene regulation in Salmonella typhimurium.Regulation of oxidative stress response by CosR, an essential response regulator in Campylobacter jejuni.Salmonella enterica serovar typhimurium induces cell death in bovine monocyte-derived macrophages by early sipB-dependent and delayed sipB-independent mechanismsConstruction and characterization of genetically defined aro omp mutants of enterotoxigenic Escherichia coli and preliminary studies of safety and immunogenicity in humans.In vivo genetic analysis indicates that PhoP-PhoQ and the Salmonella pathogenicity island 2 type III secretion system contribute independently to Salmonella enterica serovar Typhimurium virulence.Cloning vectors and fluorescent proteins can significantly inhibit Salmonella enterica virulence in both epithelial cells and macrophages: implications for bacterial pathogenesis studies.Identification of new secreted effectors in Salmonella enterica serovar TyphimuriumNegative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid.HLA-B27 modulates intracellular growth of Salmonella pathogenicity island 2 mutants and production of cytokines in infected monocytic U937 cells.The inner membrane histidine kinase EnvZ senses osmolality via helix-coil transitions in the cytoplasm.Transcription of the SsrAB regulon is repressed by alkaline pH and is independent of PhoPQ and magnesium concentration.
P2860
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P2860
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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@zh-my
2000年学术文章
@zh-sg
2000年學術文章
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name
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@en
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@nl
type
label
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@en
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@nl
prefLabel
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@en
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@nl
P2093
P2860
P1476
OmpR regulates the two-component system SsrA-ssrB in Salmonella pathogenicity island 2.
@en
P2093
P2860
P304
P356
10.1128/JB.182.3.771-781.2000
P407
P577
2000-02-01T00:00:00Z