Nitric oxide formation in acutely rejecting cardiac allografts correlates with GTP cyclohydrolase I activity.
about
Deficient BH4 production via de novo and salvage pathways regulates NO responses to cytokines in adult cardiac myocytesSepiapterin decreases acute rejection and apoptosis in cardiac transplants independently of changes in nitric oxide and inducible nitric-oxide synthase dimerization.Tetrahydrobiopterin in the prevention of hypertonia in hypoxic fetal brain.Cardiac myocyte-specific overexpression of human GTP cyclohydrolase I protects against acute cardiac allograft rejectionThe complex role of iNOS in acutely rejecting cardiac transplants.Reactive oxygen and reactive nitrogen as signaling molecules for caspase 3 activation in acute cardiac transplant rejection.
P2860
Nitric oxide formation in acutely rejecting cardiac allografts correlates with GTP cyclohydrolase I activity.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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name
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@en
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@nl
type
label
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@en
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@nl
prefLabel
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@en
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@nl
P2093
P2860
P356
P1433
P1476
Nitric oxide formation in acut ...... GTP cyclohydrolase I activity.
@en
P2093
Ashwani K Khanna
Gail Hilton
Galen M Pieper
Jeannette Vásquez-Vivar
Nadine L N Halligan
Vani Nilakantan
P2860
P304
P356
10.1042/BJ20050575
P407
P577
2005-11-01T00:00:00Z