Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response - Wikidata - awgldk - TriplyDB

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

http://www.wikidata.org/entity/Q39512596

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Protein Interferon-Stimulated Gene 15 Conjugation Delays but Does Not Overcome Coronavirus Proliferation in a Model of Fulminant Hepatitis Accessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Autocrine interferon priming in macrophages but not dendritic cells results in enhanced cytokine and chemokine production after coronavirus infection Cell-type-specific type I interferon antagonism influences organ tropism of murine coronavirus The Nucleocapsid Protein of Coronaviruses Acts as a Viral Suppressor of RNA Silencing in Mammalian Cells Protective role of Toll-like Receptor 3-induced type I interferon in murine coronavirus infection of macrophages.Brain Invasion by Mouse Hepatitis Virus Depends on Impairment of Tight Junctions and Beta Interferon Production in Brain Microvascular Endothelial Cells.Lung epithelial cells have virus-specific and shared gene expression responses to infection by diverse respiratory viruses.Cell-type-specific activation of the oligoadenylate synthetase-RNase L pathway by a murine coronavirus.Post-transcriptional control of type I interferon induction by porcine reproductive and respiratory syndrome virus in its natural host cells.Nucleocytoplasmic transport of nucleocapsid proteins of enveloped RNA viruses.Cellular RNA Helicase DDX1 Is Involved in Transmissible Gastroenteritis Virus nsp14-Induced Interferon-Beta Production Visualizing coronavirus RNA synthesis in time by using click chemistry.Computational and Experimental Studies of ADP-Ribosylation.Murine coronavirus delays expression of a subset of interferon-stimulated genes Antagonism of the interferon-induced OAS-RNase L pathway by murine coronavirus ns2 protein is required for virus replication and liver pathology.Murine hepatitis virus nsp14 exoribonuclease activity is required for resistance to innate immunity.Porcine reproductive and respiratory syndrome virus suppresses post-transcriptionally the protein expression of IFN-β by upregulating cellular microRNAs in porcine alveolar macrophages in vitro.Alpha/Beta Interferon (IFN-α/β) Signaling in Astrocytes Mediates Protection against Viral Encephalomyelitis and Regulates IFN-γ-Dependent Responses.

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Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

http://www.wikidata.org/entity/Q39512596

description

2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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2009年论文

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name

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

@en

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response.

@nl

type

label

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

@en

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response.

@nl

prefLabel

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

@en

Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response.

@nl

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Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon Response

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Kristine M Rose

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P2860

Severe acute respiratory syndrome coronavirus-like virus in Chinese horseshoe bats

AKAP18 contains a phosphoesterase domain that binds AMP

Structural and functional basis for ADP-ribose and poly(ADP-ribose) binding by viral macro domains

Croup is associated with the novel coronavirus NL63

Processing of Genome 5′ Termini as a Strategy of Negative-Strand RNA Viruses to Avoid RIG-I-Dependent Interferon Induction

Interferon Regulatory Factor IRF-7 Induces the Antiviral Alpha Interferon Response and Protects against Lethal West Nile Virus Infection

Murine Coronavirus Mouse Hepatitis Virus Is Recognized by MDA5 and Induces Type I Interferon in Brain Macrophages/Microglia

Negative regulation of cytoplasmic RNA-mediated antiviral signaling

The nsP3 macro domain is important for Sindbis virus replication in neurons and neurovirulence in mice

Crystal structures of the X-domains of a Group-1 and a Group-3 coronavirus reveal that ADP-ribose-binding may not be a conserved property

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689-712

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scholarly article

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10.3390/V1030689

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3

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20221421

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2835314

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