Genetic loss of HCN1 channels is exciting, but is it epileptic? - Wikidata - awgldk - TriplyDB

Genetic loss of HCN1 channels is exciting, but is it epileptic?

Genetic loss of HCN1 channels is exciting, but is it epileptic?

http://www.wikidata.org/entity/Q39553828

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Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potential HCN1 Gain-Of-Function Mutations - A New Cause of Epileptic Encephalopathy.Nitric oxide selectively suppresses IH currents mediated by HCN1-containing channels.

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Genetic loss of HCN1 channels is exciting, but is it epileptic?

http://www.wikidata.org/entity/Q39553828

description

2010 nî lūn-bûn

@nan

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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2010年论文

@zh

2010年论文

@zh-cn

name

Genetic loss of HCN1 channels is exciting, but is it epileptic?

@en

Genetic loss of HCN1 channels is exciting, but is it epileptic?

@nl

type

label

Genetic loss of HCN1 channels is exciting, but is it epileptic?

@en

Genetic loss of HCN1 channels is exciting, but is it epileptic?

@nl

prefLabel

Genetic loss of HCN1 channels is exciting, but is it epileptic?

@en

Genetic loss of HCN1 channels is exciting, but is it epileptic?

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J.1535-7511.2009.01352.X

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Epilepsy Currents

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Genetic loss of HCN1 channels is exciting, but is it epileptic?

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Nicholas P Poolos

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The VGL-Chanome: A Protein Superfamily Specialized for Electrical Signaling and Ionic Homeostasis

Kv4.2 knockout mice demonstrate increased susceptibility to convulsant stimulation

Loss of dendritic HCN1 subunits enhances cortical excitability and epileptogenesis.

Progressive dendritic HCN channelopathy during epileptogenesis in the rat pilocarpine model of epilepsy.

Epileptogenic channelopathies: experimental models of human pathologies.

Absence epilepsy and sinus dysrhythmia in mice lacking the pacemaker channel HCN2

The Yin and Yang of the H-Channel and Its Role in Epilepsy.

A behavioral role for dendritic integration: HCN1 channels constrain spatial memory and plasticity at inputs to distal dendrites of CA1 pyramidal neurons.

h channel-dependent deficit of theta oscillation resonance and phase shift in temporal lobe epilepsy.

Mouse models of human KCNQ2 and KCNQ3 mutations for benign familial neonatal convulsions show seizures and neuronal plasticity without synaptic reorganization.

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49-51

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