Interaction between 24-hydroxycholesterol, oxidative stress, and amyloid-β in amplifying neuronal damage in Alzheimer's disease: three partners in crime.
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Exercise-induced cognitive plasticity, implications for mild cognitive impairment and Alzheimer's diseaseOxysterols in the pathogenesis of major chronic diseasesOxidized cholesterol as the driving force behind the development of Alzheimer's diseaseLoading into nanoparticles improves quercetin's efficacy in preventing neuroinflammation induced by oxysterolsVitiligo: a possible model of degenerative diseases.Beta-amyloid peptides enhance the proliferative response of activated CD4CD28 lymphocytes from Alzheimer disease patients and from healthy elderly.Steady-state levels of retinal 24S-hydroxycholesterol are maintained by glial cells intervention after elevation of intraocular pressure in the rat.Neuronal DNA damage response-associated dysregulation of signalling pathways and cholesterol metabolism at the earliest stages of Alzheimer-type pathologyDietary lipids and their oxidized products in Alzheimer's disease.Amyloid-β production: major link between oxidative stress and BACE1.The link between altered cholesterol metabolism and Alzheimer's disease.NADPH oxidase and neurodegeneration24(S)-Hydroxycholesterol as a Modulator of Neuronal Signaling and Survival.Protein engineering of microbial cholesterol oxidases: a molecular approach toward development of new enzymes with new properties.Current Knowledge about Oxysterols: A Review.Oxidized LDL lipids increase β-amyloid production by SH-SY5Y cells through glutathione depletion and lipid raft formation.The enzyme lecithin-cholesterol acyltransferase esterifies cerebrosterol and limits the toxic effect of this oxysterol on SH-SY5Y cells.Up-regulation of β-amyloidogenesis in neuron-like human cells by both 24- and 27-hydroxycholesterol: protective effect of N-acetyl-cysteine.Fatty acid transporting proteins: Roles in brain development, aging, and stroke.Aβ1-42-mediated down-regulation of Uch-L1 is dependent on NF-κB activation and impaired BACE1 lysosomal degradation.24(S)-hydroxycholesterol induces neuronal cell death through necroptosis, a form of programmed necrosis.Amyloidogenicity and toxicity of the reverse and scrambled variants of amyloid-β 1-42Cholesterol-metabolizing enzyme cytochrome P450 46A1 as a pharmacologic target for Alzheimer's disease.Vitamin E supplementation modulates the biological effects of omega-3 fatty acids in naturally aged rats.The level of 24-Hydroxycholesteryl Esters is an Early Marker of Alzheimer's Disease.Does serum uric acid act as a modulator of cerebrospinal fluid Alzheimer's disease biomarker related cognitive decline?A silver lining for 24-hydroxycholesterol in Alzheimer's disease: The involvement of the neuroprotective enzyme sirtuin 1.Strikingly different effects of cholesterol and 7-ketocholesterol on lipid bilayer-mediated aggregation of amyloid beta (1-42).
P2860
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P2860
Interaction between 24-hydroxycholesterol, oxidative stress, and amyloid-β in amplifying neuronal damage in Alzheimer's disease: three partners in crime.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Interaction between 24-hydroxy ...... ease: three partners in crime.
@en
Interaction between 24-hydroxy ...... ease: three partners in crime.
@nl
type
label
Interaction between 24-hydroxy ...... ease: three partners in crime.
@en
Interaction between 24-hydroxy ...... ease: three partners in crime.
@nl
prefLabel
Interaction between 24-hydroxy ...... ease: three partners in crime.
@en
Interaction between 24-hydroxy ...... ease: three partners in crime.
@nl
P2093
P2860
P50
P1433
P1476
Interaction between 24-hydroxy ...... sease: three partners in crime
@en
P2093
Barbara Sottero
Elena Tamagno
Fiorella Biasi
Giuseppe Poli
Michela Guglielmotto
Simona Gargiulo
P2860
P304
P356
10.1111/J.1474-9726.2011.00681.X
P577
2011-04-05T00:00:00Z