Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
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Macrophage Expression of Inflammatory Genes in Response to EMCV InfectionPhysical and functional interaction between Hck tyrosine kinase and guanine nucleotide exchange factor C3G results in apoptosis, which is independent of C3G catalytic domainSrc family kinases participate in the regulation of encephalomyocarditis virus-induced cyclooxygenase-2 expression by macrophagesViruses cause type 1 diabetes in animals.The diverse functions of Src family kinases in macrophages.Effect of p38 mitogen-activated protein kinase on the replication of encephalomyocarditis virusRole of vav1- and src-related tyrosine kinases in macrophage activation by CpG DNA.
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Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@en
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@nl
type
label
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@en
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@nl
prefLabel
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@en
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice.
@nl
P2093
P2860
P1433
P1476
Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice
@en
P2093
P2860
P304
P356
10.1128/JVI.75.4.1949-1957.2001
P407
P50
P577
2001-02-01T00:00:00Z