Competition amongst Eph receptors regulates contact inhibition of locomotion and invasiveness in prostate cancer cells.
about
Collective cell migration in developmentEphA receptor signaling--complexity and emerging themesEphrin-Eph signaling in embryonic tissue separationTumor cell migration in complex microenvironmentsProtein interacting with C kinase 1 suppresses invasion and anchorage-independent growth of astrocytic tumor cellsMyosin 1b functions as an effector of EphB signaling to control cell repulsionVariable combinations of specific ephrin ligand/Eph receptor pairs control embryonic tissue separationDirectional collective cell migration emerges as a property of cell interactionsContact inhibition of locomotion determines cell-cell and cell-substrate forces in tissuesCadherin-11 mediates contact inhibition of locomotion during Xenopus neural crest cell migrationA role for PP1/NIPP1 in steering migration of human cancer cellsStructurally encoded intraclass differences in EphA clusters drive distinct cell responsesIntegrins in the Spotlight of CancerProtein kinase A can block EphA2 receptor-mediated cell repulsion by increasing EphA2 S897 phosphorylationInter-Cellular Forces Orchestrate Contact Inhibition of LocomotionForcing contact inhibition of locomotionMicrotubule remodelling is required for the front-rear polarity switch during contact inhibition of locomotionContact repulsion controls the dispersion and final distribution of Cajal-Retzius cells.EphA2 cleavage by MT1-MMP triggers single cancer cell invasion via homotypic cell repulsion.Contact inhibition of locomotion probabilities drive solitary versus collective cell migration.Interplay between chemotaxis and contact inhibition of locomotion determines exploratory cell migrationDynamic interactions between cancer cells and the embryonic microenvironment regulate cell invasion and reveal EphB6 as a metastasis suppressor.Phosphorylation of Rab-coupling protein by LMTK3 controls Rab14-dependent EphA2 trafficking to promote cell:cell repulsion.Front-Rear Polarization by Mechanical Cues: From Single Cells to Tissues.The differential expression of EphB2 and EphB4 receptor kinases in normal bladder and in transitional cell carcinoma of the bladderA small molecule agonist of EphA2 receptor tyrosine kinase inhibits tumor cell migration in vitro and prostate cancer metastasis in vivo.Attenuation of eph receptor kinase activation in cancer cells by coexpressed ephrin ligandsDesign, synthesis and biological evaluation of novel bromophenol derivatives incorporating indolin-2-one moiety as potential anticancer agents.Eph receptors and ephrins as targets for cancer therapy.A disalicylic acid-furanyl derivative inhibits ephrin binding to a subset of Eph receptorsThe tumour-promoting receptor tyrosine kinase, EphB4, regulates expression of integrin-β8 in prostate cancer cells.The Cell Adhesion Molecule Necl-4/CADM4 Serves as a Novel Regulator for Contact Inhibition of Cell Movement and Proliferation.Eph receptor function is modulated by heterooligomerization of A and B type Eph receptorsMolecular mechanisms of cell segregation and boundary formation in development and tumorigenesis.The role of EphB4 and IGF-IR expression in breast cancer cellsCadherin Switch during EMT in Neural Crest Cells Leads to Contact Inhibition of Locomotion via Repolarization of ForcesGlobal evaluation of Eph receptors and ephrins in lung adenocarcinomas identifies EphA4 as an inhibitor of cell migration and invasion.Ligand-independent activation of EphA2 by arachidonic acid induces metastasis-like behaviour in prostate cancer cells.Profiling Eph receptor expression in cells and tissues: a targeted mass spectrometry approachThe multifaceted roles of Eph/ephrin signaling in breast cancer.
P2860
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P2860
Competition amongst Eph receptors regulates contact inhibition of locomotion and invasiveness in prostate cancer cells.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Competition amongst Eph recept ...... ness in prostate cancer cells.
@en
Competition amongst Eph recept ...... ness in prostate cancer cells.
@nl
type
label
Competition amongst Eph recept ...... ness in prostate cancer cells.
@en
Competition amongst Eph recept ...... ness in prostate cancer cells.
@nl
prefLabel
Competition amongst Eph recept ...... ness in prostate cancer cells.
@en
Competition amongst Eph recept ...... ness in prostate cancer cells.
@nl
P2093
P356
P1433
P1476
Competition amongst Eph recept ...... ness in prostate cancer cells.
@en
P2093
Catherine D Nobes
David Gillatt
Jennifer Batson
Jessica Charlet
Jon D Oxley
Jonathan W Astin
Raj A Persad
Shereen Kadir
P2888
P304
P356
10.1038/NCB2122
P577
2010-11-14T00:00:00Z
P6179
1008430021