Aryl hydrocarbon receptor nuclear translocator/hypoxia-inducible factor-1{beta} plays a critical role in maintaining glucose-stimulated anaplerosis and insulin release from pancreatic {beta}-cells.
about
The possible mechanisms underlying the impairment of HIF-1α pathway signaling in hyperglycemia and the beneficial effects of certain therapiesMetabolomics applied to the pancreatic isletIntronic cis-regulatory modules mediate tissue-specific and microbial control of angptl4/fiaf transcriptionThe potential for chemical mixtures from the environment to enable the cancer hallmark of sustained proliferative signallingMitochondrial metabolism of pyruvate is essential for regulating glucose-stimulated insulin secretionSilencing of hypoxia-inducible factor-1β induces anti-tumor effects in hepatoma cell lines under tumor hypoxia.Suppression of the hypoxia inducible factor-1 function by redistributing the aryl hydrocarbon receptor nuclear translocator from nucleus to cytoplasm.Molecular network-based analysis of guizhi-shaoyao-zhimu decoction, a TCM herbal formula, for treatment of diabetic peripheral neuropathy.Decreased serum glucose and glycosylated hemoglobin levels in patients with Chuvash polycythemia: a role for HIF in glucose metabolismToxicological function of adipose tissue: focus on persistent organic pollutants.Mass spectrometry-based metabolomics towards understanding of gene functions with a diversity of biological contexts.Role of prolyl hydroxylase domain proteins in the regulation of insulin secretion.Volatile anesthetics suppress glucose-stimulated insulin secretion in MIN6 cells by inhibiting glucose-induced activation of hypoxia-inducible factor 1.The Rab-GTPase activating protein, TBC1D1, is critical for maintaining normal glucose homeostasis and β-cell mass.Metabolomic analysis of pancreatic β-cell insulin release in response to glucose.Deletion of ARNT/HIF1β in pancreatic beta cells does not impair glucose homeostasis in mice, but is associated with defective glucose sensing ex vivo.Diabetes - the Role of Metabolomics in the Discovery of New Mechanisms and Novel Biomarkers
P2860
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P2860
Aryl hydrocarbon receptor nuclear translocator/hypoxia-inducible factor-1{beta} plays a critical role in maintaining glucose-stimulated anaplerosis and insulin release from pancreatic {beta}-cells.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@en
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@nl
type
label
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@en
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@nl
prefLabel
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@en
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@nl
P2093
P2860
P356
P1476
Aryl hydrocarbon receptor nucl ...... from pancreatic {beta}-cells.
@en
P2093
Jamie W Joseph
Peter Huypens
Renjitha Pillai
Shawn D Wettig
Stephanie Schaefer
Tanya Sheinin
P2860
P304
P356
10.1074/JBC.M110.149062
P407
P577
2010-11-08T00:00:00Z