Granulocyte-macrophage colony-stimulating factor- and tumor necrosis factor alpha-mediated matrix metalloproteinase production by human osteoblasts and monocytes after infection with Brucella abortus.
about
Ex vivo innate immune cytokine signature of enhanced risk of relapsing brucellosisPotential role of fibroblast-like synoviocytes in joint damage induced by Brucella abortus infection through production and induction of matrix metalloproteinases.Brucella abortus invasion of osteoblasts inhibits bone formation.The Effector Protein BPE005 from Brucella abortus Induces Collagen Deposition and Matrix Metalloproteinase 9 Downmodulation via Transforming Growth Factor β1 in Hepatic Stellate Cells.Brucella abortus induces TNF-α-dependent astroglial MMP-9 secretion through mitogen-activated protein kinasesTNF-α induces matrix metalloproteinase-9-dependent soluble intercellular adhesion molecule-1 release via TRAF2-mediated MAPKs and NF-κB activation in osteoblast-like MC3T3-E1 cells.Internal affairs: investigating the Brucella intracellular lifestyle.Excretory/secretory products from two Fasciola hepatica isolates induce different transcriptional changes and IL-10 release in LPS-activated bovine "BOMA" macrophages.Brucella abortus Invasion of Osteocytes Modulates Connexin 43 and Integrin Expression and Induces Osteoclastogenesis via Receptor Activator of NF-κB Ligand and Tumor Necrosis Factor Alpha SecretionPathogenic potential of Escherichia coli clinical strains from orthopedic implant infections towards human osteoblastic cells.Brucella and Osteoarticular Cell Activation: Partners in Crime.TLR2 and AP-1/NF-kappaB are involved in the regulation of MMP-9 elicited by heat killed Listeria monocytogenes in human monocytic THP-1 cells.Toll-like receptor 6 plays an important role in host innate resistance to Brucella abortus infection in miceBrucella abortus invasion of synoviocytes inhibits apoptosis and induces bone resorption through RANKL expression.Brucella abortus induces intracellular retention of MHC-I molecules in human macrophages down-modulating cytotoxic CD8(+) T cell responses.Macrophage-elicited osteoclastogenesis in response to Brucella abortus infection requires TLR2/MyD88-dependent TNF-α production.Brucella abortus Promotes a Fibrotic Phenotype in Hepatic Stellate Cells, with Concomitant Activation of the Autophagy Pathway.Inhibition of Osteoblast Function by Brucella abortus is Reversed by Dehydroepiandrosterone and Involves ERK1/2 and Estrogen Receptor.
P2860
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P2860
Granulocyte-macrophage colony-stimulating factor- and tumor necrosis factor alpha-mediated matrix metalloproteinase production by human osteoblasts and monocytes after infection with Brucella abortus.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@en
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@nl
type
label
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@en
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@nl
prefLabel
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@en
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@nl
P2093
P2860
P356
P1476
Granulocyte-macrophage colony- ...... fection with Brucella abortus.
@en
P2093
Guillermo H Giambartolomei
M Victoria Delpino
Paula Barrionuevo
Romina Scian
P2860
P304
P356
10.1128/IAI.00934-10
P407
P577
2010-10-18T00:00:00Z