Functional analysis of the Helicobacter pylori cag pathogenicity island reveals both VirD4-CagA-dependent and VirD4-CagA-independent mechanisms.
about
Helicobacter pylori and mitogen-activated protein kinases mediate activator protein-1 (AP-1) subcomponent protein expression and DNA-binding activity in gastric epithelial cells.Quantitative analysis of representative proteome components and clustering of Helicobacter pylori clinical strains.NF-kappaB activation by Helicobacter pylori requires Akt-mediated phosphorylation of p65Roles of the plasticity regions of Helicobacter pylori in gastroduodenal pathogenesis.Comparison of Helicobacter pylori virulence gene expression in vitro and in the Rhesus macaque.Helicobacter pylori exploits a unique repertoire of type IV secretion system components for pilus assembly at the bacteria-host cell interfaceCagI is an essential component of the Helicobacter pylori Cag type IV secretion system and forms a complex with CagL.Pathogenicity islands in bacterial pathogenesisThe versatile bacterial type IV secretion systems.Cag pathogenicity island-specific responses of gastric epithelial cells to Helicobacter pylori infectionMolecular microbiology and pathogenesis of Helicobacter and Campylobacter updated: a meeting report of the 11th conference on Campylobacter, Helicobacter and related organisms.Genomic investigation into strain heterogeneity and pathogenic potential of the emerging gastrointestinal pathogen Campylobacter ureolyticus.Copper promotes TFF1-mediated Helicobacter pylori colonization.Relation of CagA seropositivity to cagPAI phenotype and histological grade of gastritis in patients with Helicobacter pylori infection.Deletion of cagA gene of Helicobacter pylori by PCR products.Duodenal ulcer promoting gene of Helicobacter pyloriFunctional and intracellular signaling differences associated with the Helicobacter pylori AlpAB adhesin from Western and East Asian strains.Clinical relevance of cagPAI intactness in Helicobacter pylori isolates from Vietnam.Helicobacter pylori AlpA and AlpB bind host laminin and influence gastric inflammation in gerbils.Show me the substrates: modulation of host cell function by type IV secretion systems.Helicobacter pylori interferes with an embryonic stem cell micro RNA cluster to block cell cycle progression.Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancer.The outs and ins of bacterial type IV secretion substrates.Roles of virD4 and cagG genes in the cag pathogenicity island of Helicobacter pylori using a Mongolian gerbil modelAssembly and mechanisms of bacterial type IV secretion machines.c-Src and c-Abl kinases control hierarchic phosphorylation and function of the CagA effector protein in Western and East Asian Helicobacter pylori strainsH. pylori infection: bacterial virulence factors and cytokine gene polymorphisms as determinants of infection outcome.Heterogeneity in the activity of Mexican Helicobacter pylori strains in gastric epithelial cells and its association with diversity in the cagA gene.Expression of cagA, virB/D Complex and/or vacA Genes in Helicobacter pylori Strains Originating from Patients with Gastric DiseasesPeptidomimetic Small Molecules Disrupt Type IV Secretion System Activity in Diverse Bacterial Pathogens.Gene distribution of cagII in Helicobacter pylori-infected patients of Zhejiang Province.An RGD helper sequence in CagL of Helicobacter pylori assists in interactions with integrins and injection of CagACorrelation between cag pathogenicity island composition and Helicobacter pylori-associated gastroduodenal diseaseThe beta1 integrin activates JNK independent of CagA, and JNK activation is required for Helicobacter pylori CagA+-induced motility of gastric cancer cellsThe Helicobacter pylori cytotoxin CagA is essential for suppressing host heat shock protein expression.Prevalence of vacA, cagA and babA2 genes in Cuban Helicobacter pylori isolates.Selective upregulation of endothelial E-selectin in response to Helicobacter pylori-induced gastritis.Roles of the Putative Type IV-like Secretion System Key Component VirD4 and PrsA in Pathogenesis of Streptococcus suis Type 2.Characterization of monospecies biofilm formation by Helicobacter pylori.Assembly and molecular mode of action of the Helicobacter pylori Cag type IV secretion apparatus.
P2860
Q30441110-AA932B52-61A4-4C4D-92AD-320E48D46CB5Q33262632-6F3C54DE-D7AC-4B35-9167-DA7F51271DCCQ33409108-A1DFA861-6FEC-42AA-970A-306115A38DDEQ33708486-85150120-2115-48CC-A7B0-2C65E3B3DAC1Q33946722-1A293D62-82FA-4D46-A9EA-D68AF83D0CFAQ34016256-FF7373DA-22A1-4754-9FDC-7A140C14A152Q34228625-12EF5E15-F3AC-4457-9F94-2FCC4332E9ABQ34290188-95718525-7534-4B94-BD13-BDF11B1AB84EQ34307587-60BF7EA9-5C81-40AC-B644-664A9300D3BFQ34381529-4A1C087E-BF5E-491D-8B48-75E0DC957B4DQ34722995-46D5B672-F3A6-4EC3-AAAA-751F69AB4B4BQ34983496-BA469720-1B6E-42E5-89F0-A9E58DA14DD3Q35043831-B2367867-E94C-45E8-874B-F75AE9BA065DQ35045503-8D4BCD13-27B9-4C87-B346-E77952EC9B91Q35045616-E1421A6A-801F-4E8C-91FB-67A2DBA831E0Q35087438-D2D56AE3-013A-4561-8D5A-F427EF4251D5Q35087443-AE8EB64C-389B-4B20-BB62-9111AA132034Q35109774-8FD3057F-1E5C-48EF-A081-A16A37D0E933Q35139355-6BA1876C-934A-42AA-8B57-FE355C2B95E4Q35142697-15783A6A-B401-43B9-B7F7-6D7E27C65535Q35540614-246BFA13-8C85-4354-88BB-21C1687703C9Q35550724-94C13739-D54E-476F-85FE-C56EAD6EC379Q35579496-61A79CC3-AB88-471F-9016-787AE6A4F515Q35597383-4ADB034D-83D2-4E5A-BF9C-0004C968AAE2Q35814707-7A18DE2D-5794-4C5F-B32A-DBEAC81F3E1BQ35858125-8B837426-C83A-495A-A470-4B45F1A9194FQ35861502-F23C723A-3725-4206-9831-CE1947DC5E00Q35913569-BCAE1AD4-894E-4F5A-8C74-868F6028BE54Q35920514-E627731B-35B4-4E61-B1EC-52A0B2E99C85Q36000469-B1E83FC3-5186-4832-8F51-9C3B5C0E92D0Q36066207-8AB178D9-AEBF-417F-9535-06FAA60BC4BFQ36159090-7199BAB8-E9B6-4500-953C-D031840248ACQ36376644-B3E55F2E-40A1-449B-BA64-F60DBB777171Q36646335-CE54D41F-E74A-4198-9987-1A14539E7DDEQ36814315-FF8BC461-625D-48BB-8DD6-4B3B1A90312FQ37122179-050D0FF0-3CE5-4691-8DDC-48D54A5D2715Q37256471-7A977113-0D32-45C1-8B16-F2B04B7515BFQ37463701-02CE131A-B452-442E-B7B1-70A722527803Q37734935-63FF99D8-480C-4DFE-AD3F-156AC5DDBBB2Q37847237-7D107A85-5DD3-4D36-A75B-B9A9BC02647F
P2860
Functional analysis of the Helicobacter pylori cag pathogenicity island reveals both VirD4-CagA-dependent and VirD4-CagA-independent mechanisms.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@en
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@nl
type
label
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@en
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@nl
prefLabel
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@en
Functional analysis of the Hel ...... 4-CagA-independent mechanisms.
@nl
P2093
P2860
P1476
Functional analysis of the Hel ...... D4-CagA-independent mechanisms
@en
P2093
Matthias Selbach
Stefan Moese
Steffen Backert
P2860
P304
P356
10.1128/IAI.70.2.665-671.2002
P407
P577
2002-02-01T00:00:00Z