The epidermal growth factor receptor modulates DNA double-strand break repair by regulating non-homologous end-joining.
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MicroRNA and signal transduction pathways in tumor radiation responseEpidermal growth factor receptor targeted nuclear delivery and high-resolution whole cell X-ray imaging of Fe3O4@TiO2 nanoparticles in cancer cells.XRCC3 and RAD51 expression are associated with clinical factors in breast cancer.High mobility group A1 protein expression reduces the sensitivity of colon and thyroid cancer cells to antineoplastic drugsTargeting DDX3 with a small molecule inhibitor for lung cancer therapy.Dual Targeting of Akt and mTORC1 Impairs Repair of DNA Double-Strand Breaks and Increases Radiation Sensitivity of Human Tumor Cells.Early tumor progression associated with enhanced EGFR signaling with bortezomib, cetuximab, and radiotherapy for head and neck cancer.DNA Damage Response Assessments in Human Tumor Samples Provide Functional Biomarkers of Radiosensitivity.Geminin prevents DNA damage in vagal neural crest cells to ensure normal enteric neurogenesisSPOC1 modulates DNA repair by regulating key determinants of chromatin compaction and DNA damage response.Epidermal growth factor regulates hematopoietic regeneration after radiation injuryRapid Diminution in the Level and Activity of DNA-Dependent Protein Kinase in Cancer Cells by a Reactive Nitro-Benzoxadiazole CompoundInsulin-like growth factor binding protein-3 (IGFBP-3): Novel ligands mediate unexpected functions.Cytokines in radiobiological responses: a review.Strategies for optimizing the response of cancer and normal tissues to radiation.Dual targeting of PI3K and MEK enhances the radiation response of K-RAS mutated non-small cell lung cancer.Potential use of HMG-CoA reductase inhibitors (statins) as radioprotective agents.Beyond ATM: the protein kinase landscape of the DNA damage response.Reversion of the ErbB malignant phenotype and the DNA damage response.DNA double-strand break repair as determinant of cellular radiosensitivity to killing and target in radiation therapy.Low-fidelity compensatory backup alternative DNA repair pathways may unify current carcinogenesis theories.Molecular targeting in combination with platinum-based chemoradiotherapy in head and neck cancer treatment.EGF receptor inhibition radiosensitizes NSCLC cells by inducing senescence in cells sustaining DNA double-strand breaks.Molecular Targeting of Growth Factor Receptor Signaling in Radiation Oncology.Enhanced dependency of KRAS-mutant colorectal cancer cells on RAD51-dependent homologous recombination repair identified from genetic interactions in Saccharomyces cerevisiae.Harnessing the power of proteomics for identification of oncogenic, druggable signalling pathways in cancer.p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation.MRE11 and ATM AKTivate pro-survival signaling.Influence of allelic Variations of hypoxia-related and DNA repair genes on patient outcome and toxicity in head and neck cancer treated with radiotherapy plus cetuximab.Protein tyrosine phosphatase (PTP) inhibition enhances chromosomal stability after genotoxic stress: decreased chromosomal instability (CIN) at the expense of enhanced genomic instability (GIN)?EGFR Mutations Compromise Hypoxia-Associated Radiation Resistance through Impaired Replication Fork-Associated DNA Damage Repair.Involvement of the insulin-like growth factor binding proteins in the cancer cell response to DNA damage.Delayed cell death associated with mitotic catastrophe in γ-irradiated stem-like glioma cells.Epidermal Growth Factor and Granulocyte Colony Stimulating Factor Signaling Are Synergistic for Hematopoietic Regeneration.Oligosaccharide G19 inhibits U-87 MG human glioma cells growth in vitro and in vivo by targeting epidermal growth factor (EGF) and activating p53/p21 signaling.Analyzing the influence of kinase inhibitors on DNA repair by differential proteomics of chromatin-interacting proteins and nuclear phospho-proteins.Icotinib hydrochloride enhances chemo- and radiosensitivity by inhibiting EGFR signaling and attenuating RAD51 expression and function in Hela S3 cells.DNA Repair.EGFR ligands and DNA repair genes: genomic predictors of complete response after capecitabine-based chemoradiotherapy in locally advanced rectal cancer.NF-κB inhibition by dimethylaminoparthenolide radiosensitizes non-small-cell lung carcinoma by blocking DNA double-strand break repair.
P2860
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P2860
The epidermal growth factor receptor modulates DNA double-strand break repair by regulating non-homologous end-joining.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
The epidermal growth factor re ...... ng non-homologous end-joining.
@en
The epidermal growth factor re ...... ng non-homologous end-joining.
@nl
type
label
The epidermal growth factor re ...... ng non-homologous end-joining.
@en
The epidermal growth factor re ...... ng non-homologous end-joining.
@nl
prefLabel
The epidermal growth factor re ...... ng non-homologous end-joining.
@en
The epidermal growth factor re ...... ng non-homologous end-joining.
@nl
P2093
P1433
P1476
The epidermal growth factor re ...... ng non-homologous end-joining.
@en
P2093
Ekkehard Dikomey
Jarob Saker
Jochen Dahm-Daphi
Katharina Holst
Malte Kriegs
Thorsten Rieckmann
Ulla Kasten-Pisula
P304
P356
10.1016/J.DNAREP.2010.05.005
P577
2010-06-19T00:00:00Z