Essential role of Gas6 for glomerular injury in nephrotoxic nephritis. - Wikidata - awgldk - TriplyDB

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

http://www.wikidata.org/entity/Q39738129

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Structural basis for Gas6-Axl signalling.The clearance of dying cells: table for two Phagocytosis of apoptotic cells in homeostasis Familial multiple coagulation factor deficiencies: new biologic insight from rare genetic bleeding disorders Uterine sensitization-associated gene-1 (USAG-1), a novel BMP antagonist expressed in the kidney, accelerates tubular injury.Gas6 and the receptor tyrosine kinase Axl in clear cell renal cell carcinoma.GAS6 enhances repair following cuprizone-induced demyelination.Immunobiology of the TAM receptors.Axl tyrosine kinase protects against tubulo-interstitial apoptosis and progression of renal failure in a murine model of chronic kidney disease and hyperphosphataemia.Loss of Growth arrest specific gene 6 (Gas6) results in altered GnRH neuron migration, delayed vaginal opening and sexual maturation in mice Dual involvement of growth arrest-specific gene 6 in the early phase of human IgA nephropathy Plasma concentrations of Gas6 and sAxl correlate with disease activity in systemic lupus erythematosus.Renal gene and protein expression signatures for prediction of kidney disease progression Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease.Growth arrest-specific protein 6 plasma concentrations during septic shock.Growth arrest-specific gene 6 (Gas6) levels are elevated in patients with chronic renal failure.Circulating growth arrest-specific protein 6 levels are associated with erythropoietin resistance in hemodialysis patients.Shp2 Associates with and Enhances Nephrin Tyrosine Phosphorylation and Is Necessary for Foot Process Spreading in Mouse Models of Podocyte Injury.Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathy TAM receptor signaling and autoimmune disease.Increased Mer and Axl receptor tyrosine kinase expression on glomeruli in lupus nephritis.Human vitamin K-dependent GAS6: gene structure, allelic variation, and association with stroke.Opposing Roles of Tyrosine Kinase Receptors Mer and Axl Determine Clinical Outcomes in Experimental Immune-Mediated Nephritis.Conditional Deletion of Smad1 Ameliorates Glomerular Injury in Progressive Glomerulonephritis Inhibition of histone deacetylase activates side population cells in kidney and partially reverses chronic renal injury.A protective role of Mer receptor tyrosine kinase in nephrotoxic serum-induced nephritis.Intracellular signaling pathways involved in Gas6-Axl-mediated survival of endothelial cells.Expression and activation of STAT3 in chronic proliferative immune complex glomerulonephritis and the effect of fosinopril.Serum growth arrest-specific protein 6 levels are a reliable biomarker of disease activity in systemic lupus erythematosus.Plasma growth arrest-specific protein 6 levels in premenopausal and postmenopausal women: the role of endogenous estrogen.Induction of Gas6 protein in CCl4-induced rat liver injury and anti-apoptotic effect on hepatic stellate cells.Gas6 deficiency prevents liver inflammation, steatohepatitis, and fibrosis in mice.Nephrin is necessary for podocyte recovery following injury in an adult mature glomerulus.

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P2860

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

http://www.wikidata.org/entity/Q39738129

description

2002 nî lūn-bûn

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2002年の論文

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年学术文章

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2002年學術文章

@yue

2002年學術文章

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2002年學術文章

@zh-hant

name

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@en

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@nl

type

label

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@en

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@nl

prefLabel

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@en

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis.

@nl

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P1433

Journal of Clinical Investigation

P1476

Essential role of Gas6 for glomerular injury in nephrotoxic nephritis

@en

P2093

Atsushi Fukatsu

http://www.w3.org/2001/XMLSchema#string

Hidenori Arai

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Kojiro Nagai

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Motoko Yanagita

http://www.w3.org/2001/XMLSchema#string

Toru Kita

http://www.w3.org/2001/XMLSchema#string

Toru Nakano

http://www.w3.org/2001/XMLSchema#string

Toshio Doi

http://www.w3.org/2001/XMLSchema#string

Tsuyoshi Ito

http://www.w3.org/2001/XMLSchema#string

Yoshikazu Ishimoto

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P2860

The protein encoded by a growth arrest-specific gene (gas6) is a new member of the vitamin K-dependent proteins related to protein S, a negative coregulator in the blood coagulation cascade

Gas6 regulates mesangial cell proliferation through Axl in experimental glomerulonephritis

Expression of axl, a transforming receptor tyrosine kinase, in normal and malignant hematopoiesis

Signaling through the ARK tyrosine kinase receptor protects from apoptosis in the absence of growth stimulation

Site-directed mutagenesis by gene targeting in mouse embryo-derived stem cells

Aminopeptidase A: a nephritogenic target antigen of nephrotoxic serum.

Deficiency or inhibition of Gas6 causes platelet dysfunction and protects mice against thrombosis.

P-selectin deficiency exacerbates experimental glomerulonephritis: a protective role for endothelial P-selectin in inflammation.

Lack of chemokine receptor CCR1 enhances Th1 responses and glomerular injury during nephrotoxic nephritis.

Fibrinogen mediates platelet-polymorphonuclear leukocyte cooperation during immune-complex glomerulonephritis in rats

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239-246

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10.1172/JCI0214861

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10.1172/JCI14861

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2

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12122116

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151046

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