Inducible costimulator is essential for collagen-induced arthritis. - Wikidata - awgldk - TriplyDB

Inducible costimulator is essential for collagen-induced arthritis.

Inducible costimulator is essential for collagen-induced arthritis.

http://www.wikidata.org/entity/Q39741100

about

Biology of recently discovered cytokines: interleukin-17--a unique inflammatory cytokine with roles in bone biology and arthritis Distinct functional motifs within the IL-17 receptor regulate signal transduction and target gene expression Ageing, autoimmunity and arthritis: Perturbations of TCR signal transduction pathways with ageing - a biochemical paradigm for the ageing immune system IL-23 drives a pathogenic T cell population that induces autoimmune inflammation ICOS promotes IL-17 synthesis in colonic intraepithelial lymphocytes in IL-10-/- mice.B7h is required for T cell activation, differentiation, and effector function.Candida-elicited murine Th17 cells express high Ctla-4 compared with Th1 cells and are resistant to costimulation blockade Targeting the Human T-Cell Inducible COStimulator Molecule with a Monoclonal Antibody Prevents Graft-vs-Host Disease and Preserves Graft vs Leukemia in a Xenograft Murine Model Attenuation of immune-mediated influenza pneumonia by targeting the inducible co-stimulator (ICOS) molecule on T cells Selective upregulation of microRNA expression in peripheral blood leukocytes in IL-10-/- mice precedes expression in the colon Evidence for ligand-independent multimerization of the IL-17 receptor.Activation of OX40 augments Th17 cytokine expression and antigen-specific uveitis.Expression and function of inducible co-stimulator in patients with systemic lupus erythematosus: possible involvement in excessive interferon-gamma and anti-double-stranded DNA antibody production.Th17 down-regulation is involved in reduced progression of schistosomiasis fibrosis in ICOSL KO mice.B7RP-1-ICOS interactions are required for optimal infection-induced expansion of CD4+ Th1 and Th2 responses IL-10 signaling in CD4+ T cells is critical for the pathogenesis of collagen-induced arthritis.Molecular targets in immune-mediated diseases: focus on rheumatoid arthritis.Novel treatments for systemic lupus erythematosus.Prostacyclin-IP signaling and prostaglandin E2-EP2/EP4 signaling both mediate joint inflammation in mouse collagen-induced arthritis.The phenotype of circulating follicular-helper T cells in patients with rheumatoid arthritis defines CD200 as a potential therapeutic target.In vivo equilibrium of proinflammatory IL-17+ and regulatory IL-10+ Foxp3+ RORgamma t+ T cells T-cell co-stimulatory pathways in autoimmunity.An overview of IL-17 function and signaling.Interleukin-23/Th17 pathways and inflammatory bowel disease.Regulatory T-cell vaccination independent of auto-antigen.Targeting lymphocyte co-stimulation: from bench to bedside.Pharmacotherapy: concepts of pathogenesis and emerging treatments. Co-stimulation and T cells as therapeutic targets.Treatment of systemic lupus erythematosus: new advances in targeted therapy.Targeting co-stimulatory pathways: transplantation and autoimmunity.Cytokines link osteoblasts and inflammation: microarray analysis of interleukin-17- and TNF-alpha-induced genes in bone cells.Follicular helper T cell in immunity and autoimmunity Synovial fluid proteome in rheumatoid arthritis.The role of the ICOS-B7h T cell costimulatory pathway in transplantation immunity.A Rac1 inhibitory peptide suppresses antibody production and paw swelling in the murine collagen-induced arthritis model of rheumatoid arthritis TLR9-dependent IL-23/IL-17 is required for the generation of Stachybotrys chartarum-induced hypersensitivity pneumonitis.Sustained T cell Rap1 signaling is protective in the collagen-induced arthritis model of rheumatoid arthritis.CD4(+) T-cell activation is differentially modulated by bacteria-primed dendritic cells, but is generally down-regulated by n-3 polyunsaturated fatty acids

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P2860

Inducible costimulator is essential for collagen-induced arthritis.

http://www.wikidata.org/entity/Q39741100

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

Inducible costimulator is essential for collagen-induced arthritis.

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Inducible costimulator is essential for collagen-induced arthritis.

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type

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Inducible costimulator is essential for collagen-induced arthritis.

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Inducible costimulator is essential for collagen-induced arthritis.

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prefLabel

Inducible costimulator is essential for collagen-induced arthritis.

@en

Inducible costimulator is essential for collagen-induced arthritis.

@nl

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P1433

Journal of Clinical Investigation

P1476

Inducible costimulator is essential for collagen-induced arthritis.

@en

P2093

Chen Dong

http://www.w3.org/2001/XMLSchema#string

Julie Duong

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Piper Treuting

http://www.w3.org/2001/XMLSchema#string

Richard A Flavell

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Roza I Nurieva

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P2860

ICOS is an inducible T-cell co-stimulator structurally and functionally related to CD28

Human IL-17: a novel cytokine derived from T cells

IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis

T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

T-cell co-stimulation through B7RP-1 and ICOS

ICOS co-stimulatory receptor is essential for T-cell activation and function

ICOS is critical for CD40-mediated antibody class switching

Differential T cell costimulatory requirements in CD28-deficient mice

ICOS is essential for effective T-helper-cell responses

B7h, a novel costimulatory homolog of B7.1 and B7.2, is induced by TNFalpha.

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701-706

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10.1172/JCI17321

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5

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111

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2003-03-01T00:00:00Z

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151904

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