Calcineurin splicing variant calcineurin Aβ1 improves cardiac function after myocardial infarction without inducing hypertrophy.
about
Muscle lim protein isoform negatively regulates striated muscle actin dynamics and differentiationCounteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein PhosphatasesOngoing controversies surrounding cardiac remodeling: is it black and white-or rather fifty shades of gray?Wrecked regulation of intrinsically disordered proteins in diseases: pathogenicity of deregulated regulatorsThe alternative heart: impact of alternative splicing in heart disease.Calcineurin suppresses AMPK-dependent cytoprotective autophagy in cardiomyocytes under oxidative stress.Cardiac CaM Kinase II genes δ and γ contribute to adverse remodeling but redundantly inhibit calcineurin-induced myocardial hypertrophy.Cardiac mTOR rescues the detrimental effects of diet-induced obesity in the heart after ischemia-reperfusion.Understanding cardiovascular disease: a journey through the genome (and what we found there).Myocyte signalling in leucocyte recruitment to the heart.Intron retention in mRNA: No longer nonsense: Known and putative roles of intron retention in normal and disease biology.Emerging roles for RNA-binding proteins as effectors and regulators of cardiovascular disease.Calcineurin signaling in the heart: The importance of time and place.Extensive alternative splicing transitions during postnatal skeletal muscle development are required for calcium handling functionsThe unique C terminus of the calcineurin isoform CNAβ1 confers non-canonical regulation of enzyme activity by Ca2+ and calmodulin.Follistatin-like 3 mediates paracrine fibroblast activation by cardiomyocytes.Guidelines for translational research in heart failure.Intron retention in viruses and cellular genes: Detention, border controls and passports.Induction of the calcineurin variant CnAβ1 after myocardial infarction reduces post-infarction ventricular remodelling by promoting infarct vascularization
P2860
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P2860
Calcineurin splicing variant calcineurin Aβ1 improves cardiac function after myocardial infarction without inducing hypertrophy.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Calcineurin splicing variant c ...... without inducing hypertrophy.
@en
Calcineurin splicing variant c ...... without inducing hypertrophy.
@nl
type
label
Calcineurin splicing variant c ...... without inducing hypertrophy.
@en
Calcineurin splicing variant c ...... without inducing hypertrophy.
@nl
prefLabel
Calcineurin splicing variant c ...... without inducing hypertrophy.
@en
Calcineurin splicing variant c ...... without inducing hypertrophy.
@nl
P2093
P50
P1433
P1476
Calcineurin splicing variant c ...... n without inducing hypertrophy
@en
P2093
Jesús M Gómez-Salinero
Ken Suzuki
Kunihiko Takahashi
Marina M López-Olañeta
Padmini Sarathchandra
Renée Germack
Takuya Narita
Yasunori Shintani
P304
P356
10.1161/CIRCULATIONAHA.110.012211
P407
P577
2011-05-31T00:00:00Z