about
Telethonin deficiency is associated with maladaptation to biomechanical stress in the mammalian heartMyocardial expression of the arginine:glycine amidinotransferase gene is elevated in heart failure and normalized after recovery: potential implications for local creatine synthesisHeterogeneic nature of adult cardiac side population cells.Choice of cell-delivery route for skeletal myoblast transplantation for treating post-infarction chronic heart failure in rat.Heat shock protein-27 delays acute rejection after cardiac transplantation: an experimental model.Regulated inositol-requiring protein 1-dependent decay as a mechanism of corin RNA and protein deficiency in advanced human systolic heart failure.Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function.Integrated allelic, transcriptional, and phenomic dissection of the cardiac effects of titin truncations in health and diseaseExpression of follistatin-related genes is altered in heart failure.Myocardial insulin-like growth factor-I gene expression during recovery from heart failure after combined left ventricular assist device and clenbuterol therapy.Expression of human membrane skeleton protein genes for protein 4.1 and betaIISigma2-spectrin assayed by real-time RT-PCR.Calcineurin splicing variant calcineurin Aβ1 improves cardiac function after myocardial infarction without inducing hypertrophy.Phenylephrine requires the TATA box to activate transcription of GLUT1 in neonatal rat cardiac myocytes.A gene expression profile of the myocardial response to clenbuterol.Follistatin-like 3 mediates paracrine fibroblast activation by cardiomyocytes.Overexpression of the transcription factor Hand1 causes predisposition towards arrhythmia in mice.Direct intramyocardial but not intracoronary injection of bone marrow cells induces ventricular arrhythmias in a rat chronic ischemic heart failure model.A quantitative gene expression profile of matrix metalloproteinases (MMPS) and their inhibitors (TIMPS) in the myocardium of patients with deteriorating heart failure requiring left ventricular assist device support.Molecular signature of recovery following combination left ventricular assist device (LVAD) support and pharmacologic therapy.Increased toll-like receptor 4 in the myocardium of patients requiring left ventricular assist devices.Expression of extracellular matrix genes during myocardial recovery from heart failure after left ventricular assist device support.Increased expression of extracellular matrix regulators TIMP1 and MMP1 in deteriorating heart failure.Reverse remodelling and recovery from heart failure are associated with complex patterns of gene expression.Sequential expression of three known protective genes in cardiac biopsies after transplantation.Correlation between vascular responsivensss and expression of novel transcripts of the ETA-receptor in human vascular tissueQuantitative myocardial cytokine expression and activation of the apoptotic pathway in patients who require left ventricular assist devices
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Leanne E Felkin
@ast
Leanne E Felkin
@en
Leanne E Felkin
@es
Leanne E Felkin
@nl
Leanne E Felkin
@sl
type
label
Leanne E Felkin
@ast
Leanne E Felkin
@en
Leanne E Felkin
@es
Leanne E Felkin
@nl
Leanne E Felkin
@sl
prefLabel
Leanne E Felkin
@ast
Leanne E Felkin
@en
Leanne E Felkin
@es
Leanne E Felkin
@nl
Leanne E Felkin
@sl
P106
P21
P31
P496
0000-0003-1402-8314
P569
2000-01-01T00:00:00Z