Endocrine disruptor, dioxin (TCDD)-induced mitochondrial dysfunction and apoptosis in human trophoblast-like JAR cells.
about
The placenta in toxicology. Part IV: Battery of toxicological test systems based on human placentaIntegrating mitochondriomics in children's environmental healthHealth and cellular impacts of air pollutants: from cytoprotection to cytotoxicityTCDD induces the hypoxia-inducible factor (HIF)-1α regulatory pathway in human trophoblastic JAR cellsCausal effects of synthetic chemicals on mitochondrial deficits and diabetes pandemic.Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical lawsMitochondrial metabolism and diabetesSwainsonine induces apoptosis through mitochondrial pathway and caspase activation in goat trophoblasts.2, 3, 7, 8-Tetrachlorodibenzo-P-dioxin (TCDD) induces premature senescence in human and rodent neuronal cells via ROS-dependent mechanisms2,3,7,8-Tetrachlorodibenzo-p-dioxin-mediated production of reactive oxygen species is an essential step in the mechanism of action to accelerate human keratinocyte differentiation.Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte FunctionsDioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis-persistent pollutants affect programmed cell death.'Omics' and endocrine-disrupting chemicals - new paths forward.Nonylphenol-induced apoptotic cell death in mouse TM4 Sertoli cells via the generation of reactive oxygen species and activation of the ERK signaling pathway.Novel cell-based assay reveals associations of circulating serum AhR-ligands with metabolic syndrome and mitochondrial dysfunction.Effect of transient TCDD exposure on immortalized human trophoblast-derived cell lines.Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants.Mitochondrial dysfunction induced by Bisphenol A is a factor of its hepatotoxicity in rats.Actein alleviates 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated cellular dysfunction in osteoblastic MC3T3-E1 cells.In Vitro Evaluation of Mitochondrial Function and Estrogen Signaling in Cell Lines Exposed to the Antiseptic Cetylpyridinium Chloride.Butyl paraben promotes apoptosis in human trophoblast cells through increased oxidative stress-induced endoplasmic reticulum stress.2, 3, 7, 8-Tetrachlorodibenzo-p-dioxin promotes endothelial cell apoptosis through activation of EP3/p38MAPK/Bcl-2 pathway.Olive oil and its phenolic compounds (hydroxytyrosol and tyrosol) ameliorated TCDD-induced heptotoxicity in rats via inhibition of oxidative stress and apoptosis.Antioxidant compounds in the seaweed Gelidiella acerosa protects human Peripheral Blood Mononuclear Cells against TCDD induced toxicity.
P2860
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P2860
Endocrine disruptor, dioxin (TCDD)-induced mitochondrial dysfunction and apoptosis in human trophoblast-like JAR cells.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
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name
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@en
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@nl
type
label
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@en
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@nl
prefLabel
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@en
Endocrine disruptor, dioxin (T ...... an trophoblast-like JAR cells.
@nl
P2093
P2860
P356
P1476
Endocrine disruptor, dioxin (T ...... man trophoblast-like JAR cells
@en
P2093
Chii-Reuy Tzeng
Su-Chee Chen
Tien-Ling Liao
Yau-Huei Wei
P2860
P304
P356
10.1093/MOLEHR/GAQ004
P50
P577
2010-01-18T00:00:00Z